In Situ Reprogrammings of Splenic CD11b+ Cells by Nano‐Hypoxia to Promote Inflamed Damage Site‐Specific Angiogenesis (Adv. Funct. Mater. 32/2023)

Splenic Filtering of Nanoparticles In article number 2302817, Hak‐Joon Sung, Seung Eun Yu, and co‐workers show that the inevitable splenic filtering of nanoparticles can be turned into an advantage. By loading a hypoxia‐mimetic agent onto liposomes, splenic CD11b+ cells undergo vasculogenic reprogra...

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Veröffentlicht in:Advanced functional materials 2023-08, Vol.33 (32), p.n/a
Hauptverfasser: Chung, Seyong, Kim, Si Yeong, Lee, Kyubae, Baek, Sewoom, Ha, Hyun‐Su, Kim, Dae‐Hyun, Park, Suji, Lee, Chan Hee, Kim, Hye‐Seon, Shin, Young Min, Yu, Seung Eun, Sung, Hak‐Joon
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container_end_page n/a
container_issue 32
container_start_page
container_title Advanced functional materials
container_volume 33
creator Chung, Seyong
Kim, Si Yeong
Lee, Kyubae
Baek, Sewoom
Ha, Hyun‐Su
Kim, Dae‐Hyun
Park, Suji
Lee, Chan Hee
Kim, Hye‐Seon
Shin, Young Min
Yu, Seung Eun
Sung, Hak‐Joon
description Splenic Filtering of Nanoparticles In article number 2302817, Hak‐Joon Sung, Seung Eun Yu, and co‐workers show that the inevitable splenic filtering of nanoparticles can be turned into an advantage. By loading a hypoxia‐mimetic agent onto liposomes, splenic CD11b+ cells undergo vasculogenic reprogramming, making them home inflamed sites and rescue damaged tissues under ischemia or resection.
doi_str_mv 10.1002/adfm.202370196
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subjects angiogenesis
Damage
Filtration
Hypoxia
ischemia
Materials science
Nanoparticles
spleen
title In Situ Reprogrammings of Splenic CD11b+ Cells by Nano‐Hypoxia to Promote Inflamed Damage Site‐Specific Angiogenesis (Adv. Funct. Mater. 32/2023)
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