The secondary FLT3-ITD F691L mutation induces resistance to AC220 in FLT3-ITD+ AML but retains in vitro sensitivity to PKC412 and Sunitinib

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Bibliographische Detailangaben
Veröffentlicht in:	Leukemia 2013-06, Vol.27 (6), p.1416-1418
Hauptverfasser:	Albers, C, Leischner, H, Verbeek, M, Yu, C, Illert, A L, Peschel, C, von Bubnoff, N, Duyster, J
Format:	Artikel
Sprache:	eng
Schlagworte:	692/699/67/1059/2326 692/699/67/1990/283/1897 692/699/67/68 Antigens Antineoplastic Agents - therapeutic use Benzothiazoles - therapeutic use Blood Bone marrow Cancer Research Cancer therapies Chemotherapy Critical Care Medicine Cytokines Drug Resistance, Neoplasm - genetics Female fms-Like Tyrosine Kinase 3 - genetics fms-Like Tyrosine Kinase 3 - physiology Hematology Humans Indoles - therapeutic use Intensive Internal Medicine letter-to-the-editor Letters to the editor Leukemia Leukemia, Myeloid, Acute - drug therapy Leukemia, Myeloid, Acute - genetics Leukocytes Ligands Medicine Medicine & Public Health Middle Aged Mutation Oncology Phenylurea Compounds - therapeutic use Pyrroles - therapeutic use Remission (Medicine) Staurosporine - analogs & derivatives Staurosporine - therapeutic use Stem cells Transplants & implants
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