Redundant function of retinoic acid receptor isoforms in leukemogenesis unravels a prominent function of genome topology and architecture in the selection of mutagenic events in cancer
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| Veröffentlicht in: | Leukemia 2009-02, Vol.23 (2), p.417-419 |
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| container_issue | 2 |
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| container_title | Leukemia |
| container_volume | 23 |
| creator | Marinelli, A Bossi, D Pelicci, P G Minucci, S |
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| doi_str_mv | 10.1038/leu.2008.205 |
| format | Article |
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| fulltext | fulltext |
| identifier | ISSN: 0887-6924 |
| ispartof | Leukemia, 2009-02, Vol.23 (2), p.417-419 |
| issn | 0887-6924 1476-5551 |
| language | eng |
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| source | MEDLINE; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; Springer Nature - Complete Springer Journals; Nature Journals Online |
| subjects | Animals Cancer Research Cell receptors Critical Care Medicine DNA binding proteins Gene Rearrangement Genetic aspects Genomes Health aspects Hematology Intensive Internal Medicine Isoforms letter-to-the-editor Leukemia - etiology Leukemia - pathology Leukemogenesis Medicine Medicine & Public Health Mice Myelocytic leukemia Nonlymphoid leukemia Oncology Protein Isoforms Receptors, Retinoic Acid - genetics Receptors, Retinoic Acid - physiology Retinoic acid Retinoic Acid Receptor alpha Risk factors Topology |
| title | Redundant function of retinoic acid receptor isoforms in leukemogenesis unravels a prominent function of genome topology and architecture in the selection of mutagenic events in cancer |
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