Redundant function of retinoic acid receptor isoforms in leukemogenesis unravels a prominent function of genome topology and architecture in the selection of mutagenic events in cancer

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Bibliographische Detailangaben
Veröffentlicht in:	Leukemia 2009-02, Vol.23 (2), p.417-419
Hauptverfasser:	Marinelli, A, Bossi, D, Pelicci, P G, Minucci, S
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cancer Research Cell receptors Critical Care Medicine DNA binding proteins Gene Rearrangement Genetic aspects Genomes Health aspects Hematology Intensive Internal Medicine Isoforms letter-to-the-editor Leukemia - etiology Leukemia - pathology Leukemogenesis Medicine Medicine & Public Health Mice Myelocytic leukemia Nonlymphoid leukemia Oncology Protein Isoforms Receptors, Retinoic Acid - genetics Receptors, Retinoic Acid - physiology Retinoic acid Retinoic Acid Receptor alpha Risk factors Topology
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