Effects of Ca+2 channel activity on renal hemodynamics during acute attenuation of NO synthesis in the rat

Effects of Ca+2 channel activity on renal hemodynamics during acute attenuation of NO synthesis in the rat

In cultured vascular muscle cells, nitric oxide (NO) has been shown to inhibit voltage-dependent Ca2+ channels, which are involved in renal blood flow (RBF) autoregulation. Therefore, our purpose was to specify in vivo the effects of this interaction on RBF autoregulation.

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Veröffentlicht in:American journal of physiology. Renal physiology 2000-04, Vol.47 (4), p.F561
Hauptverfasser: Kramp, R A, Fourmanoir, P, Ladriere, L, Joly, E
Format: Artikel
Sprache:eng
Schlagworte:
Calcium
Hemodialysis
Kidneys
Rodents
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Zusammenfassung:In cultured vascular muscle cells, nitric oxide (NO) has been shown to inhibit voltage-dependent Ca2+ channels, which are involved in renal blood flow (RBF) autoregulation. Therefore, our purpose was to specify in vivo the effects of this interaction on RBF autoregulation.
ISSN:1931-857X
1522-1466