Portal GLP-1 administration in rats augments the insulin response to glucose via neuronal mechanisms

Balkan and Li hypothesize that the hepatic portal system may constitute one site of GLP-1 action. They investigate whether an indirect mechanism triggered in the hepatoportal circulatory system may be involved in the stimulation of insulin secretion by endogenously secreted GLP-1.

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Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Regulatory, integrative and comparative physiology integrative and comparative physiology, 2000-10, Vol.48 (4), p.R1449-R1454
Hauptverfasser:	BALKAN, Börk, XUE LI
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Feeding. Feeding behavior Fundamental and applied biological sciences. Psychology Glucose Insulin Nervous system Neurons Rodents Vertebrates: anatomy and physiology, studies on body, several organs or systems
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Beschreibung
Zusammenfassung:	Balkan and Li hypothesize that the hepatic portal system may constitute one site of GLP-1 action. They investigate whether an indirect mechanism triggered in the hepatoportal circulatory system may be involved in the stimulation of insulin secretion by endogenously secreted GLP-1.
ISSN:	0363-6119 1522-1490