Effect of heat stress on LPS-induced febrile response in d-galactosamine-sensitized rats

We have previously reported that heat conditioning augments lipopolysaccharide (LPS)-induced fever in rats, which is accompanied by an accumulation of heat shock protein (HSP) in the liver and the reduction of the plasma level of tumor necrosis factor (TNF-alpha) (Kluger MJ, Rudolph K, Soszynski D,...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Regulatory, integrative and comparative physiology integrative and comparative physiology, 2001-02, Vol.49 (2), p.R338-R344
Hauptverfasser:	DOKLADNY, Karol, KOZAK, Anna, WACHULEC, Maciej, WALLEN, Erik S, MENACHE, Margaret G, KOZAK, Wieslaw, KLUGER, Matthew J, MOSELEY, Pope L
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Fever Fundamental and applied biological sciences. Psychology Heart Heat Kidneys Liver Proteins Rodents Stress Thermoregulation. Hibernation. Estivation. Ecophysiology and environmental effects Tumors Vertebrates: anatomy and physiology, studies on body, several organs or systems
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Beschreibung
Zusammenfassung:	We have previously reported that heat conditioning augments lipopolysaccharide (LPS)-induced fever in rats, which is accompanied by an accumulation of heat shock protein (HSP) in the liver and the reduction of the plasma level of tumor necrosis factor (TNF-alpha) (Kluger MJ, Rudolph K, Soszynski D, Conn CA, Leon LR, Kozak W, Wallen ES, and Moseley PL. Am J Physiol Regulatory Integrative Comp Physiol 273: R858-R863, 1997).
ISSN:	0363-6119 1522-1490