Chronic metabolic sequelae of traumatic brain injury : Prolonged suppression of somatosensory activation
Injuries to the brain acutely disrupt normal metabolic function and may deactivate functional circuits. It is unknown whether these metabolic abnormalities improve over time. We used 2-deoxyglucose (2-DG) autoradiographic image-averaging to assess local cerebral glucose utilization (lCMRGlc) of the...
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| Veröffentlicht in: | American journal of physiology. Heart and circulatory physiology 2000-09, Vol.48 (3), p.H924-H931 |
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| container_end_page | H931 |
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| container_issue | 3 |
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| container_title | American journal of physiology. Heart and circulatory physiology |
| container_volume | 48 |
| creator | PASSINEAU, M. J ZHAO, W BUSTO, R DIETRICH, W. D ALONSO, O LOOR, J. Y BRAMLETT, H. M GINSBERG, M. D |
| description | Injuries to the brain acutely disrupt normal metabolic function and may deactivate functional circuits. It is unknown whether these metabolic abnormalities improve over time. We used 2-deoxyglucose (2-DG) autoradiographic image-averaging to assess local cerebral glucose utilization (lCMRGlc) of the rat brain 2 mo after moderate (1.7-2.1 atm) fluid-percussion traumatic brain injury (FPI). |
| format | Article |
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J ; ZHAO, W ; BUSTO, R ; DIETRICH, W. D ; ALONSO, O ; LOOR, J. Y ; BRAMLETT, H. M ; GINSBERG, M. D</creator><creatorcontrib>PASSINEAU, M. J ; ZHAO, W ; BUSTO, R ; DIETRICH, W. D ; ALONSO, O ; LOOR, J. Y ; BRAMLETT, H. M ; GINSBERG, M. D</creatorcontrib><description>Injuries to the brain acutely disrupt normal metabolic function and may deactivate functional circuits. It is unknown whether these metabolic abnormalities improve over time. 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D</creatorcontrib><title>Chronic metabolic sequelae of traumatic brain injury : Prolonged suppression of somatosensory activation</title><title>American journal of physiology. Heart and circulatory physiology</title><description>Injuries to the brain acutely disrupt normal metabolic function and may deactivate functional circuits. It is unknown whether these metabolic abnormalities improve over time. We used 2-deoxyglucose (2-DG) autoradiographic image-averaging to assess local cerebral glucose utilization (lCMRGlc) of the rat brain 2 mo after moderate (1.7-2.1 atm) fluid-percussion traumatic brain injury (FPI).</description><subject>Biochemistry</subject><subject>Biological and medical sciences</subject><subject>Brain</subject><subject>Circulatory system</subject><subject>Injuries</subject><subject>Injuries of the nervous system and the skull. Diseases due to physical agents</subject><subject>Medical sciences</subject><subject>Neurology</subject><subject>Traumas. 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| identifier | ISSN: 0363-6135 |
| ispartof | American journal of physiology. Heart and circulatory physiology, 2000-09, Vol.48 (3), p.H924-H931 |
| issn | 0363-6135 1522-1539 |
| language | eng |
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| source | American Physiological Society; EZB-FREE-00999 freely available EZB journals |
| subjects | Biochemistry Biological and medical sciences Brain Circulatory system Injuries Injuries of the nervous system and the skull. Diseases due to physical agents Medical sciences Neurology Traumas. Diseases due to physical agents |
| title | Chronic metabolic sequelae of traumatic brain injury : Prolonged suppression of somatosensory activation |
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