Nitric oxide synthase inhibition reduces venous return in porcine endotoxemia

Saetre et al examined mechanisms of circulatory effects induced by nitric oxide synthase inhibition in endotoxemia in 36 pigs. Results indicate that Ng-nitro-L-arginine methyl ester may compromise cardiovascular function in endotoxemia.

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Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 1996-10, Vol.40 (4), p.H1325-H1332
Hauptverfasser:	SAETRE, T, SMISETH, O. A, SCHOLZ, T, CARLSEN, H, NORDSLETTEN, L, FAHLSTRØM, E, AASEN, A. O
Format:	Artikel
Sprache:	eng
Schlagworte:	Bacterial diseases Biological and medical sciences Cellular biology Coronary vessels Experimental bacterial diseases and models Heart Hogs Infectious diseases Medical sciences Permeability
Online-Zugang:	Volltext
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Beschreibung
Zusammenfassung:	Saetre et al examined mechanisms of circulatory effects induced by nitric oxide synthase inhibition in endotoxemia in 36 pigs. Results indicate that Ng-nitro-L-arginine methyl ester may compromise cardiovascular function in endotoxemia.
ISSN:	0363-6135 1522-1539