Endothelium-derived nitric oxide mediates hypoxic vasodilation of resistance vessels in humans

Blitzer et al tested the hypothesis that endothelium-derived nitric oxide (EDNO) contributes to hypoxia-induced vasodilation of forearm resistance vessels of healthy humans. Results support the hypothesis.

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Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 1996-09, Vol.40 (3), p.H1182-H1185
Hauptverfasser:	BLITZER, M. L, LEE, D, CREAGER, M. A
Format:	Artikel
Sprache:	eng
Schlagworte:	Biochemistry Biological and medical sciences Blood pressure Blood vessels and receptors Circulatory system Fundamental and applied biological sciences. Psychology Vertebrates: cardiovascular system
Online-Zugang:	Volltext
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Beschreibung
Zusammenfassung:	Blitzer et al tested the hypothesis that endothelium-derived nitric oxide (EDNO) contributes to hypoxia-induced vasodilation of forearm resistance vessels of healthy humans. Results support the hypothesis.
ISSN:	0363-6135 1522-1539