Proinflammatory cytokines depress cardiac efficiency by a nitric oxide-dependent mechanism

Proinflammatory cytokines (interleukin-1Beta, tumor necrosis factor-alpha, and interferon-gamma; Cytomix) depress myocardial contractile work partially by stimulating expression of inducible nitric oxide (NO) synthase (iNOS).

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Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 1998-09, Vol.44 (3), p.H1016-H1023
Hauptverfasser:	PANAS, D, KHADOUR, F. H, SZABO, C, SCHULZ, R
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Chemicals Efficiency Fundamental and applied biological sciences. Psychology Heart Vertebrates: cardiovascular system
Online-Zugang:	Volltext
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Beschreibung
Zusammenfassung:	Proinflammatory cytokines (interleukin-1Beta, tumor necrosis factor-alpha, and interferon-gamma; Cytomix) depress myocardial contractile work partially by stimulating expression of inducible nitric oxide (NO) synthase (iNOS).
ISSN:	0363-6135 1522-1539