NO contributes to neurohypophysial but not other regional cerebral fluorocarbon-induced hyperemia in cats

Wagner et al determine whether nitric oxide (NO)-mediated vasorelaxation is increased in fluorocarbon (FC)-perfused hemoglobin (Hb)-free cats because NO is not scavenged by Hb. Increases in regional cerebral blood flow after total FC-exchange transfusion are not caused by a lack of NO scavenging, wi...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 1997-10, Vol.42 (4), p.H1994-H2000
Hauptverfasser:	WAGNER, B. P, STINGELE, R, WILLIAMS, M. A, WILSON, D. A, TRAYSTMAN, R. J, HANLEY, D. F
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Cats Cerebral circulation. Blood-brain barrier. Choroid plexus. Cerebrospinal fluid. Circumventricular organ. Meninges Fundamental and applied biological sciences. Psychology Heart Hemoglobin Vertebrates: nervous system and sense organs
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Zusammenfassung:	Wagner et al determine whether nitric oxide (NO)-mediated vasorelaxation is increased in fluorocarbon (FC)-perfused hemoglobin (Hb)-free cats because NO is not scavenged by Hb. Increases in regional cerebral blood flow after total FC-exchange transfusion are not caused by a lack of NO scavenging, with the exception of neurohypophysis.
ISSN:	0363-6135 1522-1539