Selective upregulation of arterial endothelial nitric oxide synthase in pulmonary hypertension
Resta et al hypothesized that chronic hypoxia selectively upregulates endothelial nitric oxide synthase (eNOS) within the pulmonary arterial vasculature and that monocrotaline-induced pulmonary arterial hypertension selectively enhances pulmonary arterial dilation to endothelium-derived nitric oxide...
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| Veröffentlicht in: | American journal of physiology. Heart and circulatory physiology 1997-02, Vol.41 (2), p.H806-H813 |
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| container_end_page | H813 |
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| container_issue | 2 |
| container_start_page | H806 |
| container_title | American journal of physiology. Heart and circulatory physiology |
| container_volume | 41 |
| creator | RESTA, T. C GONZALES, R. J DAIL, W. G SANDERS, T. C WALKER, B. R |
| description | Resta et al hypothesized that chronic hypoxia selectively upregulates endothelial nitric oxide synthase (eNOS) within the pulmonary arterial vasculature and that monocrotaline-induced pulmonary arterial hypertension selectively enhances pulmonary arterial dilation to endothelium-derived nitric oxide-dependent dilators and upregulates arterial eNOS. |
| format | Article |
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C ; GONZALES, R. J ; DAIL, W. G ; SANDERS, T. C ; WALKER, B. R</creator><creatorcontrib>RESTA, T. C ; GONZALES, R. J ; DAIL, W. G ; SANDERS, T. C ; WALKER, B. R</creatorcontrib><description>Resta et al hypothesized that chronic hypoxia selectively upregulates endothelial nitric oxide synthase (eNOS) within the pulmonary arterial vasculature and that monocrotaline-induced pulmonary arterial hypertension selectively enhances pulmonary arterial dilation to endothelium-derived nitric oxide-dependent dilators and upregulates arterial eNOS.</description><identifier>ISSN: 0363-6135</identifier><identifier>EISSN: 1522-1539</identifier><identifier>CODEN: AJPPDI</identifier><language>eng</language><publisher>Bethesda, MD: American Physiological Society</publisher><subject>Anatomy & physiology ; Biological and medical sciences ; Blood vessels ; Blood vessels and receptors ; Enzymes ; Fundamental and applied biological sciences. 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Psychology</topic><topic>Hypertension</topic><topic>Lungs</topic><topic>Vertebrates: cardiovascular system</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>RESTA, T. C</creatorcontrib><creatorcontrib>GONZALES, R. J</creatorcontrib><creatorcontrib>DAIL, W. G</creatorcontrib><creatorcontrib>SANDERS, T. C</creatorcontrib><creatorcontrib>WALKER, B. R</creatorcontrib><collection>Pascal-Francis</collection><collection>Calcium & Calcified Tissue Abstracts</collection><collection>Chemoreception Abstracts</collection><collection>Physical Education Index</collection><collection>Toxicology Abstracts</collection><collection>Technology Research Database</collection><collection>Environmental Sciences and Pollution Management</collection><collection>Engineering Research Database</collection><collection>Biotechnology and BioEngineering Abstracts</collection><jtitle>American journal of physiology. 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| fulltext | fulltext |
| identifier | ISSN: 0363-6135 |
| ispartof | American journal of physiology. Heart and circulatory physiology, 1997-02, Vol.41 (2), p.H806-H813 |
| issn | 0363-6135 1522-1539 |
| language | eng |
| recordid | cdi_proquest_journals_229559928 |
| source | American Physiological Society; EZB-FREE-00999 freely available EZB journals; Alma/SFX Local Collection |
| subjects | Anatomy & physiology Biological and medical sciences Blood vessels Blood vessels and receptors Enzymes Fundamental and applied biological sciences. Psychology Hypertension Lungs Vertebrates: cardiovascular system |
| title | Selective upregulation of arterial endothelial nitric oxide synthase in pulmonary hypertension |
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