NO and H2O2 mechanisms of guanylate cyclase activation in oxygen-dependent responses of rat pulmonary circulation

A study was conducted to examine the role of two distinct mechanisms of soluble guanylate cyclase activation, nitric oxide and hydrogen peroxide-catalase, in the maintenance of low pulmonary artery pressures and to examine the oxygen tension responses in the isolated perfused rat lung.

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Veröffentlicht in:American journal of physiology. Lung cellular and molecular physiology 1995-04, Vol.12 (4), p.L546-L550
Hauptverfasser: MONACO, J. A, BURKE-WOLIN, T
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container_issue 4
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container_title American journal of physiology. Lung cellular and molecular physiology
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creator MONACO, J. A
BURKE-WOLIN, T
description A study was conducted to examine the role of two distinct mechanisms of soluble guanylate cyclase activation, nitric oxide and hydrogen peroxide-catalase, in the maintenance of low pulmonary artery pressures and to examine the oxygen tension responses in the isolated perfused rat lung.
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ispartof American journal of physiology. Lung cellular and molecular physiology, 1995-04, Vol.12 (4), p.L546-L550
issn 1040-0605
1522-1504
language eng
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source Alma/SFX Local Collection
subjects Air breathing
Biochemistry
Biological and medical sciences
Circulatory system
Fundamental and applied biological sciences. Psychology
Lungs
Oxygen
Respiratory system: anatomy, metabolism, gas exchange, ventilatory mechanics, respiratory hemodynamics
Rodents
Vertebrates: respiratory system
title NO and H2O2 mechanisms of guanylate cyclase activation in oxygen-dependent responses of rat pulmonary circulation
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