Asbestos exposure increases human bronchial epithelial cell fibrinolytic activity

To investigate the role of the epithelium in asbestos-induced lung injury, a cell culture model of the human airway was used. It was found that following exposure to chrysotile asbestos, epithelial cell monolayers demonstrate significant increases in their ability to digest plasma clots.

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Veröffentlicht in:American journal of physiology. Lung cellular and molecular physiology 1993-03, Vol.8 (3), p.L276-L283
Hauptverfasser: GROSS, T. J, COBB, S. M, GRUENERT, D. C, PETERSON, M. W
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container_title American journal of physiology. Lung cellular and molecular physiology
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creator GROSS, T. J
COBB, S. M
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PETERSON, M. W
description To investigate the role of the epithelium in asbestos-induced lung injury, a cell culture model of the human airway was used. It was found that following exposure to chrysotile asbestos, epithelial cell monolayers demonstrate significant increases in their ability to digest plasma clots.
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1522-1504
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subjects Asbestos
Biological and medical sciences
Chemical and industrial products toxicology. Toxic occupational diseases
Inorganic dusts (pneumoconiosises) and organic dusts (byssinosis etc.)
Lungs
Medical research
Medical sciences
Respiratory system
Toxicology
title Asbestos exposure increases human bronchial epithelial cell fibrinolytic activity
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