Asbestos exposure increases human bronchial epithelial cell fibrinolytic activity

To investigate the role of the epithelium in asbestos-induced lung injury, a cell culture model of the human airway was used. It was found that following exposure to chrysotile asbestos, epithelial cell monolayers demonstrate significant increases in their ability to digest plasma clots.

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Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Lung cellular and molecular physiology 1993-03, Vol.8 (3), p.L276-L283
Hauptverfasser:	GROSS, T. J, COBB, S. M, GRUENERT, D. C, PETERSON, M. W
Format:	Artikel
Sprache:	eng
Schlagworte:	Asbestos Biological and medical sciences Chemical and industrial products toxicology. Toxic occupational diseases Inorganic dusts (pneumoconiosises) and organic dusts (byssinosis etc.) Lungs Medical research Medical sciences Respiratory system Toxicology
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Beschreibung
Zusammenfassung:	To investigate the role of the epithelium in asbestos-induced lung injury, a cell culture model of the human airway was used. It was found that following exposure to chrysotile asbestos, epithelial cell monolayers demonstrate significant increases in their ability to digest plasma clots.
ISSN:	1040-0605 1522-1504