Hypoxia stimulates human preproendothelin-1 promoter activity in transgenic mice

The results of a study are consistent with the hypothesis that hypoxic induction of the PPET-1 gene leads to increased pulmonary production of ET-1 in diseases associated with low O2 tension.

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Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Lung cellular and molecular physiology 1997-10, Vol.17 (4), p.L848-L855
Hauptverfasser:	AVERSA, C. R, OPARIL, S, LIRA, S. A, WEBB, M. L, CARO, J, LI, H, SUN, S.-D, CHEN, Y.-F, SWERDEL, M. R, MONTICELLO, T. M, DURHAM, S. K, MINCHENKO, A
Format:	Artikel
Sprache:	eng
Schlagworte:	Biological and medical sciences Biotechnology Cellular biology Fundamental and applied biological sciences. Psychology Genes Genetic engineering Genetic technics Lungs Methods. Procedures. Technologies Oxygen Rodents Vectors (cloning, transfer, expression). Insertion sequences and transposons
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Beschreibung
Zusammenfassung:	The results of a study are consistent with the hypothesis that hypoxic induction of the PPET-1 gene leads to increased pulmonary production of ET-1 in diseases associated with low O2 tension.
ISSN:	1040-0605 1522-1504