Mouse species-specific control of hepatocarcinogenesis and metabolism by FGF19/FGF15

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Bibliographische Detailangaben
Veröffentlicht in:	Journal of hepatology 2017-06, Vol.66 (6), p.1182-1192
Hauptverfasser:	Zhou, Mei, Luo, Jian, Chen, Michael, Yang, Hong, Learned, R. Marc, DePaoli, Alex M, Tian, Hui, Ling, Lei
Format:	Artikel
Sprache:	eng
Schlagworte:	Alanine Alanine transaminase Alkaline phosphatase Animal models Animals Aspartate aminotransferase ATP Binding Cassette Transporter, Subfamily B - deficiency ATP Binding Cassette Transporter, Subfamily B - genetics ATP-Binding Cassette Sub-Family B Member 4 Bile Bile acids and salts Bile Acids and Salts - biosynthesis Biological activity Body composition Body weight Carcinogenesis - metabolism Carcinoma, hepatocellular Cholangitis Cyp7a1 Diabetes mellitus Diabetes Mellitus, Experimental - metabolism Diabetes Mellitus, Experimental - pathology Diet Drug resistance Energy expenditure FGF15 FGF19 Fibroblast growth factors Fibroblast Growth Factors - genetics Fibroblast Growth Factors - metabolism Gastroenterology and Hepatology Gene transfer Glucose Glycated Hemoglobin - metabolism Hemoglobin Hepatocellular carcinoma Homeostasis Humans Hyperglycemia - metabolism Insulin-Secreting Cells - metabolism Insulin-Secreting Cells - pathology Liver cancer Liver diseases Liver Neoplasms, Experimental - etiology Liver Neoplasms, Experimental - metabolism Male Metabolic diseases Metabolic disorders Metabolic Networks and Pathways Mice Mice, Inbred C57BL Mice, Knockout Mice, Mutant Strains Multidrug resistance Obesity Obesity - metabolism Oxygen consumption Receptors, cytoplasmic and nuclear Receptors, Cytoplasmic and Nuclear - agonists Receptors, Cytoplasmic and Nuclear - metabolism Receptors, Leptin - genetics Safety Serum levels Species Specificity STAT3 Transcription Factor - metabolism Tumorigenicity
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Beschreibung
Zusammenfassung:	Graphical abstract
ISSN:	0168-8278 1600-0641
DOI:	10.1016/j.jhep.2017.01.027