Fibrinolytic Pathways In Interleukin-17A-induced lung Injury

Fibrinolytic Pathways In Interleukin-17A-induced lung Injury

Methods Wild-type and interleukin-17A-deficient mice were exposed to 20 weeks of environmental tobacco smoke (ETS) to induce lung injury. ETS and interleukin-17A exposure caused significant increase in PAI-1 and lung injury in wild-type mice.

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Veröffentlicht in:Journal of allergy and clinical immunology 2018-02, Vol.141 (2), p.AB228-AB228
Hauptverfasser: Shetty, Sreerama, Perumal, Venkadesa, Tiwari, Nivedita, Marudamuthu, Amarnath, Kumari, Shwetha, Park, Seong, Shetty, Rashmi
Format: Artikel
Sprache:eng
Schlagworte:
Apoptosis
Cytokines
Fibrin
Inflammation
Lungs
Rodents
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Beschreibung
Zusammenfassung:Methods Wild-type and interleukin-17A-deficient mice were exposed to 20 weeks of environmental tobacco smoke (ETS) to induce lung injury. ETS and interleukin-17A exposure caused significant increase in PAI-1 and lung injury in wild-type mice.
ISSN:0091-6749
1097-6825
DOI:10.1016/j.jaci.2017.12.720