Fenofibrate Improves the Impaired Endothelial Progenitor Cell Function Through Deregulating Nalp3 Inflammasome Activity In Diabetic Mice
The current study aimed to test our hypothesis that fenofibrate improves angiogenesis and restore endothelial progenitor cell (EPC) function via deregulating NALP3 inflammasome activity in streptozotocin (STZ)-induced diabetic mice.
Gespeichert in:
Veröffentlicht in: | Clinical therapeutics 2015-08, Vol.37 (8), p.e25-e25 |
---|---|
Hauptverfasser: | , , , , , , |
Format: | Artikel |
Sprache: | eng |
Schlagworte: | |
Online-Zugang: | Volltext |
Tags: |
Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
|
container_end_page | e25 |
---|---|
container_issue | 8 |
container_start_page | e25 |
container_title | Clinical therapeutics |
container_volume | 37 |
creator | Deng, Y.-P Zhao, T Huang, F Ni, M Li, D.-J Jiang, G.-J Shen, F.-M |
description | The current study aimed to test our hypothesis that fenofibrate improves angiogenesis and restore endothelial progenitor cell (EPC) function via deregulating NALP3 inflammasome activity in streptozotocin (STZ)-induced diabetic mice. |
doi_str_mv | 10.1016/j.clinthera.2015.05.079 |
format | Article |
fullrecord | <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_journals_1693850187</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><els_id>1_s2_0_S0149291815003860</els_id><sourcerecordid>3735347581</sourcerecordid><originalsourceid>FETCH-LOGICAL-c2389-baf9e921a7d4253b7bec61beb7006e68370f2e126c0f82aef2925f2cb5530c523</originalsourceid><addsrcrecordid>eNqNkd2qEzEUhQdRsB59BgNeT81PMzO5EUrP6bFw_AGP4F3IZHba1ExSk0yhb-Bjm6Gi4JWwIWSz9kr2t6rqNcFLgknz9rjUzvp8gKiWFBO-xKVa8aRakK4VNSGrb0-rBSYrUVNBuufVi5SOGGMmOF1UP7fgg7F9VBnQbjzFcIaEitt8UTbCgO78EErDWeXQ5xj24G0OEW3AObSdvM42ePR4iGHaH9AtRNhPTmXr9-ijcieGdt44NY4qhRHQusjPNl9KF91a1UO2Gn2wGl5Wz4xyCV79Pm-qr9u7x837-uHT_W6zfqg1ZZ2oe2UECEpUO6woZ33bg25ID32LcQNNx1psKBDaaGw6qsBQQbmhuuecYc0pu6neXH3Lqj8mSFkewxR9eVKSRrCO44KtqNqrSseQUgQjT9GOKl4kwXLGLo_yD3Y5Y5e4VCvK5Po6CWWJs4Uok7bgNQwFps5yCPY_PN794zHrrFbuO1wg_f2wTFRi-WUOd86W8BJr12D2Cxy_psI</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>1693850187</pqid></control><display><type>article</type><title>Fenofibrate Improves the Impaired Endothelial Progenitor Cell Function Through Deregulating Nalp3 Inflammasome Activity In Diabetic Mice</title><source>Elsevier ScienceDirect Journals</source><source>ProQuest Central UK/Ireland</source><creator>Deng, Y.-P ; Zhao, T ; Huang, F ; Ni, M ; Li, D.-J ; Jiang, G.-J ; Shen, F.-M</creator><creatorcontrib>Deng, Y.-P ; Zhao, T ; Huang, F ; Ni, M ; Li, D.-J ; Jiang, G.-J ; Shen, F.-M</creatorcontrib><description>The current study aimed to test our hypothesis that fenofibrate improves angiogenesis and restore endothelial progenitor cell (EPC) function via deregulating NALP3 inflammasome activity in streptozotocin (STZ)-induced diabetic mice.</description><identifier>ISSN: 0149-2918</identifier><identifier>EISSN: 1879-114X</identifier><identifier>DOI: 10.1016/j.clinthera.2015.05.079</identifier><language>eng</language><publisher>Bridgewater: Elsevier Inc</publisher><subject>Amputation ; Diabetes ; Internal Medicine ; Medical Education ; Rodents ; Wound healing</subject><ispartof>Clinical therapeutics, 2015-08, Vol.37 (8), p.e25-e25</ispartof><rights>2015</rights><rights>Copyright Elsevier Limited Aug 2015</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktohtml>$$Uhttps://www.proquest.com/docview/1693850187?pq-origsite=primo$$EHTML$$P50$$Gproquest$$H</linktohtml><link.rule.ids>314,778,782,3539,27907,27908,45978,64366,64370,72220</link.rule.ids></links><search><creatorcontrib>Deng, Y.-P</creatorcontrib><creatorcontrib>Zhao, T</creatorcontrib><creatorcontrib>Huang, F</creatorcontrib><creatorcontrib>Ni, M</creatorcontrib><creatorcontrib>Li, D.-J</creatorcontrib><creatorcontrib>Jiang, G.-J</creatorcontrib><creatorcontrib>Shen, F.-M</creatorcontrib><title>Fenofibrate Improves the Impaired Endothelial Progenitor Cell Function Through Deregulating Nalp3 Inflammasome Activity In Diabetic Mice</title><title>Clinical therapeutics</title><description>The current study aimed to test our hypothesis that fenofibrate improves angiogenesis and restore endothelial progenitor cell (EPC) function via deregulating NALP3 inflammasome activity in streptozotocin (STZ)-induced diabetic mice.</description><subject>Amputation</subject><subject>Diabetes</subject><subject>Internal Medicine</subject><subject>Medical Education</subject><subject>Rodents</subject><subject>Wound healing</subject><issn>0149-2918</issn><issn>1879-114X</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2015</creationdate><recordtype>article</recordtype><sourceid>8G5</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><sourceid>DWQXO</sourceid><sourceid>GNUQQ</sourceid><sourceid>GUQSH</sourceid><sourceid>M2O</sourceid><recordid>eNqNkd2qEzEUhQdRsB59BgNeT81PMzO5EUrP6bFw_AGP4F3IZHba1ExSk0yhb-Bjm6Gi4JWwIWSz9kr2t6rqNcFLgknz9rjUzvp8gKiWFBO-xKVa8aRakK4VNSGrb0-rBSYrUVNBuufVi5SOGGMmOF1UP7fgg7F9VBnQbjzFcIaEitt8UTbCgO78EErDWeXQ5xj24G0OEW3AObSdvM42ePR4iGHaH9AtRNhPTmXr9-ijcieGdt44NY4qhRHQusjPNl9KF91a1UO2Gn2wGl5Wz4xyCV79Pm-qr9u7x837-uHT_W6zfqg1ZZ2oe2UECEpUO6woZ33bg25ID32LcQNNx1psKBDaaGw6qsBQQbmhuuecYc0pu6neXH3Lqj8mSFkewxR9eVKSRrCO44KtqNqrSseQUgQjT9GOKl4kwXLGLo_yD3Y5Y5e4VCvK5Po6CWWJs4Uok7bgNQwFps5yCPY_PN794zHrrFbuO1wg_f2wTFRi-WUOd86W8BJr12D2Cxy_psI</recordid><startdate>20150801</startdate><enddate>20150801</enddate><creator>Deng, Y.-P</creator><creator>Zhao, T</creator><creator>Huang, F</creator><creator>Ni, M</creator><creator>Li, D.-J</creator><creator>Jiang, G.-J</creator><creator>Shen, F.-M</creator><general>Elsevier Inc</general><general>Elsevier Limited</general><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7RV</scope><scope>7X7</scope><scope>7XB</scope><scope>88C</scope><scope>88E</scope><scope>8AO</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>8G5</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BENPR</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>GUQSH</scope><scope>K9.</scope><scope>KB0</scope><scope>M0S</scope><scope>M0T</scope><scope>M1P</scope><scope>M2O</scope><scope>M7N</scope><scope>MBDVC</scope><scope>NAPCQ</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>Q9U</scope></search><sort><creationdate>20150801</creationdate><title>Fenofibrate Improves the Impaired Endothelial Progenitor Cell Function Through Deregulating Nalp3 Inflammasome Activity In Diabetic Mice</title><author>Deng, Y.-P ; Zhao, T ; Huang, F ; Ni, M ; Li, D.-J ; Jiang, G.-J ; Shen, F.-M</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c2389-baf9e921a7d4253b7bec61beb7006e68370f2e126c0f82aef2925f2cb5530c523</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2015</creationdate><topic>Amputation</topic><topic>Diabetes</topic><topic>Internal Medicine</topic><topic>Medical Education</topic><topic>Rodents</topic><topic>Wound healing</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Deng, Y.-P</creatorcontrib><creatorcontrib>Zhao, T</creatorcontrib><creatorcontrib>Huang, F</creatorcontrib><creatorcontrib>Ni, M</creatorcontrib><creatorcontrib>Li, D.-J</creatorcontrib><creatorcontrib>Jiang, G.-J</creatorcontrib><creatorcontrib>Shen, F.-M</creatorcontrib><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Nursing & Allied Health Database</collection><collection>Health & Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Healthcare Administration Database (Alumni)</collection><collection>Medical Database (Alumni Edition)</collection><collection>ProQuest Pharma Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>Research Library (Alumni Edition)</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>ProQuest Central Essentials</collection><collection>ProQuest Central</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central Korea</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>Research Library Prep</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>Nursing & Allied Health Database (Alumni Edition)</collection><collection>Health & Medical Collection (Alumni Edition)</collection><collection>Healthcare Administration Database</collection><collection>Medical Database</collection><collection>Research Library</collection><collection>Algology Mycology and Protozoology Abstracts (Microbiology C)</collection><collection>Research Library (Corporate)</collection><collection>Nursing & Allied Health Premium</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><collection>ProQuest Central Basic</collection><jtitle>Clinical therapeutics</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Deng, Y.-P</au><au>Zhao, T</au><au>Huang, F</au><au>Ni, M</au><au>Li, D.-J</au><au>Jiang, G.-J</au><au>Shen, F.-M</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Fenofibrate Improves the Impaired Endothelial Progenitor Cell Function Through Deregulating Nalp3 Inflammasome Activity In Diabetic Mice</atitle><jtitle>Clinical therapeutics</jtitle><date>2015-08-01</date><risdate>2015</risdate><volume>37</volume><issue>8</issue><spage>e25</spage><epage>e25</epage><pages>e25-e25</pages><issn>0149-2918</issn><eissn>1879-114X</eissn><abstract>The current study aimed to test our hypothesis that fenofibrate improves angiogenesis and restore endothelial progenitor cell (EPC) function via deregulating NALP3 inflammasome activity in streptozotocin (STZ)-induced diabetic mice.</abstract><cop>Bridgewater</cop><pub>Elsevier Inc</pub><doi>10.1016/j.clinthera.2015.05.079</doi><oa>free_for_read</oa></addata></record> |
fulltext | fulltext |
identifier | ISSN: 0149-2918 |
ispartof | Clinical therapeutics, 2015-08, Vol.37 (8), p.e25-e25 |
issn | 0149-2918 1879-114X |
language | eng |
recordid | cdi_proquest_journals_1693850187 |
source | Elsevier ScienceDirect Journals; ProQuest Central UK/Ireland |
subjects | Amputation Diabetes Internal Medicine Medical Education Rodents Wound healing |
title | Fenofibrate Improves the Impaired Endothelial Progenitor Cell Function Through Deregulating Nalp3 Inflammasome Activity In Diabetic Mice |
url | https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-01-16T20%3A05%3A42IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=Fenofibrate%20Improves%20the%20Impaired%20Endothelial%20Progenitor%20Cell%20Function%20Through%20Deregulating%20Nalp3%20Inflammasome%20Activity%20In%20Diabetic%20Mice&rft.jtitle=Clinical%20therapeutics&rft.au=Deng,%20Y.-P&rft.date=2015-08-01&rft.volume=37&rft.issue=8&rft.spage=e25&rft.epage=e25&rft.pages=e25-e25&rft.issn=0149-2918&rft.eissn=1879-114X&rft_id=info:doi/10.1016/j.clinthera.2015.05.079&rft_dat=%3Cproquest_cross%3E3735347581%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=1693850187&rft_id=info:pmid/&rft_els_id=1_s2_0_S0149291815003860&rfr_iscdi=true |