The role of intracellular Ca2+ pool in sodium nitroprusside-induced relaxation of rat aorta smooth muscle cells

A relaxation effect of sodium nitroprusside on smooth muscle cells of rat aorta due to intracellular Ca^sup 2+^-store refilling is demonstrated using the double sucrose gap technique. It is suggested that sodium nitroprusside-induced repolarization of the smooth muscle cell membrane is associated wi...

Ausführliche Beschreibung

Gespeichert in:
Bibliographische Detailangaben
Veröffentlicht in:Bulletin of experimental biology and medicine 1999-02, Vol.127 (2), p.160-162
Hauptverfasser: Kovalev, I. V., Baskakov, M. B., Kapilevich, L. V., Medvedev, M. A., Panov, A. A., Popov, A. G.
Format: Artikel
Sprache:eng
Schlagworte:
Online-Zugang:Volltext
Tags: Tag hinzufügen
Keine Tags, Fügen Sie den ersten Tag hinzu!
container_end_page 162
container_issue 2
container_start_page 160
container_title Bulletin of experimental biology and medicine
container_volume 127
creator Kovalev, I. V.
Baskakov, M. B.
Kapilevich, L. V.
Medvedev, M. A.
Panov, A. A.
Popov, A. G.
description A relaxation effect of sodium nitroprusside on smooth muscle cells of rat aorta due to intracellular Ca^sup 2+^-store refilling is demonstrated using the double sucrose gap technique. It is suggested that sodium nitroprusside-induced repolarization of the smooth muscle cell membrane is associated with inhibition of Ca permeability and/or Ca-dependent K^sup +^ permeability of the plasma membrane.[PUBLICATION ABSTRACT]
doi_str_mv 10.1007/BF02433102
format Article
fullrecord <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_journals_1286758856</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>2889534751</sourcerecordid><originalsourceid>FETCH-LOGICAL-c189t-6d905a95e658703d127d174689002781051660ac0036354038c873091635cab73</originalsourceid><addsrcrecordid>eNpFkEtLxDAUhYMoOI5u_AUBd0r1JmkeXergqDDgZlyXmGSYDG1T8wD997aM4Opy7jl8Bw5C1wTuCYB8eFoDrRkjQE_QgnDJKkUpOUULmNyqVkqdo4uUDrMEQRYobPcOx9A5HHbYDzlq47qudDrilaZ3eAyhm_44BetLjwefYxhjSclbV_nBFuMsjq7T3zr7MMyUqDPWIWaNUx9C3uO-JDMVzOB0ic52ukvu6u8u0cf6ebt6rTbvL2-rx01liGpyJWwDXDfcCa4kMEuotETWQjUAVCoCnAgB2gAwwXgNTBklGTRkUkZ_SrZEN0fuGMNXcSm3h1DiMFW2hCohuVJcTKnbY8rEkFJ0u3aMvtfxpyXQzoO2_4OyX15FZiE</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>1286758856</pqid></control><display><type>article</type><title>The role of intracellular Ca2+ pool in sodium nitroprusside-induced relaxation of rat aorta smooth muscle cells</title><source>Springer Online Journals - JUSTICE</source><creator>Kovalev, I. V. ; Baskakov, M. B. ; Kapilevich, L. V. ; Medvedev, M. A. ; Panov, A. A. ; Popov, A. G.</creator><creatorcontrib>Kovalev, I. V. ; Baskakov, M. B. ; Kapilevich, L. V. ; Medvedev, M. A. ; Panov, A. A. ; Popov, A. G.</creatorcontrib><description>A relaxation effect of sodium nitroprusside on smooth muscle cells of rat aorta due to intracellular Ca^sup 2+^-store refilling is demonstrated using the double sucrose gap technique. It is suggested that sodium nitroprusside-induced repolarization of the smooth muscle cell membrane is associated with inhibition of Ca permeability and/or Ca-dependent K^sup +^ permeability of the plasma membrane.[PUBLICATION ABSTRACT]</description><identifier>ISSN: 0007-4888</identifier><identifier>EISSN: 1573-8221</identifier><identifier>DOI: 10.1007/BF02433102</identifier><identifier>CODEN: BEXBAN</identifier><language>eng</language><publisher>New York: Springer Nature B.V</publisher><subject>Cells ; Coronary vessels ; Muscular system ; Permeability ; Smooth muscle ; Sodium</subject><ispartof>Bulletin of experimental biology and medicine, 1999-02, Vol.127 (2), p.160-162</ispartof><rights>Kluwer Academic/Plenum Publishers 1999</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c189t-6d905a95e658703d127d174689002781051660ac0036354038c873091635cab73</citedby><cites>FETCH-LOGICAL-c189t-6d905a95e658703d127d174689002781051660ac0036354038c873091635cab73</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,776,780,27901,27902</link.rule.ids></links><search><creatorcontrib>Kovalev, I. V.</creatorcontrib><creatorcontrib>Baskakov, M. B.</creatorcontrib><creatorcontrib>Kapilevich, L. V.</creatorcontrib><creatorcontrib>Medvedev, M. A.</creatorcontrib><creatorcontrib>Panov, A. A.</creatorcontrib><creatorcontrib>Popov, A. G.</creatorcontrib><title>The role of intracellular Ca2+ pool in sodium nitroprusside-induced relaxation of rat aorta smooth muscle cells</title><title>Bulletin of experimental biology and medicine</title><description>A relaxation effect of sodium nitroprusside on smooth muscle cells of rat aorta due to intracellular Ca^sup 2+^-store refilling is demonstrated using the double sucrose gap technique. It is suggested that sodium nitroprusside-induced repolarization of the smooth muscle cell membrane is associated with inhibition of Ca permeability and/or Ca-dependent K^sup +^ permeability of the plasma membrane.[PUBLICATION ABSTRACT]</description><subject>Cells</subject><subject>Coronary vessels</subject><subject>Muscular system</subject><subject>Permeability</subject><subject>Smooth muscle</subject><subject>Sodium</subject><issn>0007-4888</issn><issn>1573-8221</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1999</creationdate><recordtype>article</recordtype><sourceid>BENPR</sourceid><recordid>eNpFkEtLxDAUhYMoOI5u_AUBd0r1JmkeXergqDDgZlyXmGSYDG1T8wD997aM4Opy7jl8Bw5C1wTuCYB8eFoDrRkjQE_QgnDJKkUpOUULmNyqVkqdo4uUDrMEQRYobPcOx9A5HHbYDzlq47qudDrilaZ3eAyhm_44BetLjwefYxhjSclbV_nBFuMsjq7T3zr7MMyUqDPWIWaNUx9C3uO-JDMVzOB0ic52ukvu6u8u0cf6ebt6rTbvL2-rx01liGpyJWwDXDfcCa4kMEuotETWQjUAVCoCnAgB2gAwwXgNTBklGTRkUkZ_SrZEN0fuGMNXcSm3h1DiMFW2hCohuVJcTKnbY8rEkFJ0u3aMvtfxpyXQzoO2_4OyX15FZiE</recordid><startdate>199902</startdate><enddate>199902</enddate><creator>Kovalev, I. V.</creator><creator>Baskakov, M. B.</creator><creator>Kapilevich, L. V.</creator><creator>Medvedev, M. A.</creator><creator>Panov, A. A.</creator><creator>Popov, A. G.</creator><general>Springer Nature B.V</general><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7X7</scope><scope>7XB</scope><scope>88A</scope><scope>88E</scope><scope>8AO</scope><scope>8FE</scope><scope>8FH</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BHPHI</scope><scope>CCPQU</scope><scope>DWQXO</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>GNUQQ</scope><scope>HCIFZ</scope><scope>K9.</scope><scope>LK8</scope><scope>M0S</scope><scope>M1P</scope><scope>M7P</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope></search><sort><creationdate>199902</creationdate><title>The role of intracellular Ca2+ pool in sodium nitroprusside-induced relaxation of rat aorta smooth muscle cells</title><author>Kovalev, I. V. ; Baskakov, M. B. ; Kapilevich, L. V. ; Medvedev, M. A. ; Panov, A. A. ; Popov, A. G.</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c189t-6d905a95e658703d127d174689002781051660ac0036354038c873091635cab73</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1999</creationdate><topic>Cells</topic><topic>Coronary vessels</topic><topic>Muscular system</topic><topic>Permeability</topic><topic>Smooth muscle</topic><topic>Sodium</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Kovalev, I. V.</creatorcontrib><creatorcontrib>Baskakov, M. B.</creatorcontrib><creatorcontrib>Kapilevich, L. V.</creatorcontrib><creatorcontrib>Medvedev, M. A.</creatorcontrib><creatorcontrib>Panov, A. A.</creatorcontrib><creatorcontrib>Popov, A. G.</creatorcontrib><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Health &amp; Medical Collection (ProQuest Medical &amp; Health Databases)</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Biology Database (Alumni Edition)</collection><collection>Medical Database (Alumni Edition)</collection><collection>ProQuest Pharma Collection</collection><collection>ProQuest SciTech Collection</collection><collection>ProQuest Natural Science Collection</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni)</collection><collection>ProQuest Central</collection><collection>ProQuest Central Essentials</collection><collection>Biological Science Collection</collection><collection>AUTh Library subscriptions: ProQuest Central</collection><collection>ProQuest Natural Science Collection</collection><collection>ProQuest One Community College</collection><collection>ProQuest Central</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>ProQuest Central Student</collection><collection>SciTech Premium Collection</collection><collection>ProQuest Health &amp; Medical Complete (Alumni)</collection><collection>Biological Sciences</collection><collection>Health &amp; Medical Collection (Alumni Edition)</collection><collection>PML(ProQuest Medical Library)</collection><collection>Biological Science Database</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><jtitle>Bulletin of experimental biology and medicine</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Kovalev, I. V.</au><au>Baskakov, M. B.</au><au>Kapilevich, L. V.</au><au>Medvedev, M. A.</au><au>Panov, A. A.</au><au>Popov, A. G.</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>The role of intracellular Ca2+ pool in sodium nitroprusside-induced relaxation of rat aorta smooth muscle cells</atitle><jtitle>Bulletin of experimental biology and medicine</jtitle><date>1999-02</date><risdate>1999</risdate><volume>127</volume><issue>2</issue><spage>160</spage><epage>162</epage><pages>160-162</pages><issn>0007-4888</issn><eissn>1573-8221</eissn><coden>BEXBAN</coden><abstract>A relaxation effect of sodium nitroprusside on smooth muscle cells of rat aorta due to intracellular Ca^sup 2+^-store refilling is demonstrated using the double sucrose gap technique. It is suggested that sodium nitroprusside-induced repolarization of the smooth muscle cell membrane is associated with inhibition of Ca permeability and/or Ca-dependent K^sup +^ permeability of the plasma membrane.[PUBLICATION ABSTRACT]</abstract><cop>New York</cop><pub>Springer Nature B.V</pub><doi>10.1007/BF02433102</doi><tpages>3</tpages></addata></record>
fulltext fulltext
identifier ISSN: 0007-4888
ispartof Bulletin of experimental biology and medicine, 1999-02, Vol.127 (2), p.160-162
issn 0007-4888
1573-8221
language eng
recordid cdi_proquest_journals_1286758856
source Springer Online Journals - JUSTICE
subjects Cells
Coronary vessels
Muscular system
Permeability
Smooth muscle
Sodium
title The role of intracellular Ca2+ pool in sodium nitroprusside-induced relaxation of rat aorta smooth muscle cells
url https://sfx.bib-bvb.de/sfx_tum?ctx_ver=Z39.88-2004&ctx_enc=info:ofi/enc:UTF-8&ctx_tim=2025-02-01T08%3A30%3A35IST&url_ver=Z39.88-2004&url_ctx_fmt=infofi/fmt:kev:mtx:ctx&rfr_id=info:sid/primo.exlibrisgroup.com:primo3-Article-proquest_cross&rft_val_fmt=info:ofi/fmt:kev:mtx:journal&rft.genre=article&rft.atitle=The%20role%20of%20intracellular%20Ca2+%20pool%20in%20sodium%20nitroprusside-induced%20relaxation%20of%20rat%20aorta%20smooth%20muscle%20cells&rft.jtitle=Bulletin%20of%20experimental%20biology%20and%20medicine&rft.au=Kovalev,%20I.%20V.&rft.date=1999-02&rft.volume=127&rft.issue=2&rft.spage=160&rft.epage=162&rft.pages=160-162&rft.issn=0007-4888&rft.eissn=1573-8221&rft.coden=BEXBAN&rft_id=info:doi/10.1007/BF02433102&rft_dat=%3Cproquest_cross%3E2889534751%3C/proquest_cross%3E%3Curl%3E%3C/url%3E&disable_directlink=true&sfx.directlink=off&sfx.report_link=0&rft_id=info:oai/&rft_pqid=1286758856&rft_id=info:pmid/&rfr_iscdi=true