Delayed clinical decompensation and death after pediatric nifedipine overdose

On admission, 5 hours after ingestion, his vital signs were heart rate, 150 to 170 beats/min; blood pressure, 100/40 mm Hg; respiratory rate, 36 to 44 breaths/min; and oxygen saturation, 97% to 100% on room air. Subtle evidence of toxicity (resting tachycardia, bradycardia, mild hypotension, or hype...

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Veröffentlicht in:	The American journal of emergency medicine 2007-02, Vol.25 (2), p.197-198
Hauptverfasser:	Miller, Michael A., MD, Masneri, David A., DO, Herold, Thomas, MD
Format:	Artikel
Sprache:	eng
Schlagworte:	Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy Biological and medical sciences Blood pressure Calcium Channel Blockers - poisoning Cardiac arrhythmia Child, Preschool Drug intoxications. Doping Drug Overdose Emergency Emergency and intensive care: neonates and children. Prematurity. Sudden death Emergency medical care Fatal Outcome Heart Failure - chemically induced Heart rate Humans Hyperglycemia Ingestion Intensive care medicine Male Medical sciences Medical treatment Nifedipine - poisoning Overdose Pediatrics Pharmacology. Drug treatments Time Factors Toxicity
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Zusammenfassung:	On admission, 5 hours after ingestion, his vital signs were heart rate, 150 to 170 beats/min; blood pressure, 100/40 mm Hg; respiratory rate, 36 to 44 breaths/min; and oxygen saturation, 97% to 100% on room air. Subtle evidence of toxicity (resting tachycardia, bradycardia, mild hypotension, or hyperglycemia) should prompt extreme caution and perhaps provoke aggressive clinical intervention and extended observation in nifedipine overdose.
ISSN:	0735-6757 1532-8171
DOI:	10.1016/j.ajem.2006.04.015