Hypercaloric diet models do not develop heart failure, but the excess sucrose promotes contractility dysfunction

Several diseases are associated with excess of adipose tissue, and obesity is considered an independent risk factor for the development of cardiac remodeling and heart failure. Dietary aspects have been studied to elucidate the mechanisms involved in these processes. Thus, the purpose was the develo...

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Veröffentlicht in:	PloS one 2020-02, Vol.15 (2), p.e0228860
Hauptverfasser:	Martins Matias, Amanda, Murucci Coelho, Priscila, Bermond Marques, Vinícius, Dos Santos, Leonardo, Monteiro de Assis, Aricia Leone Evangelista, Valentim Nogueira, Breno, Lima-Leopoldo, Ana Paula, Soares Leopoldo, André
Format:	Artikel
Sprache:	eng
Schlagworte:	Adipose tissue Analysis Animal models Animals Atria Atrium Biology and Life Sciences Blood pressure Calcium (intracellular) Calcium ions Calcium Signaling Carbohydrates Cardiac function Cardiomyocytes Characterization Collagen Collagen - metabolism Comorbidity Congestive heart failure Diet Diet, Carbohydrate Loading - adverse effects Diet, High-Fat - adverse effects Dietary Fats - administration & dosage Dietary Fats - adverse effects Dietary Sucrose - administration & dosage Dietary Sucrose - adverse effects Disease Models, Animal Diseases Energy Intake Health sciences Heart Heart failure Heart Failure - etiology Heart Failure - pathology Heart Failure - physiopathology Heart hypertrophy Hemodynamics Hypertrophy Insulin Laboratory animals Male Mathematical models Medicine and Health Sciences Metabolism Models, Cardiovascular Morphology Muscle contraction Myocardial Contraction Myocardium - metabolism Myocardium - pathology Myocytes, Cardiac - physiology Nutrition research Obesity Obesity - complications Obesity - pathology Obesity - physiopathology Ostomy Parameters Physical education Physical Sciences Physiology Rats Rats, Wistar Risk analysis Risk factors Sucrose Sugar Tibia Ventricle Ventricular Remodeling - physiology
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