Enhanced Disease Severity in Helicobacter pylori-Infected Mice Deficient in Fas Signaling

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Veröffentlicht in:	Infection and Immunity 2002-05, Vol.70 (5), p.2591-2597
Hauptverfasser:	JONES, Nicola L, DAY, Andrew S, JENNINGS, Hilary, SHANNON, Patrick T, GALINDO-MATA, Esther, SHERMAN, Philip M
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apoptosis Atrophy Bacteriology Biological and medical sciences Fas antigen Fas Ligand Protein fas Receptor - physiology Female Fundamental and applied biological sciences. Psychology Gastric Mucosa - pathology gld gene Helicobacter Infections - complications Helicobacter Infections - immunology Helicobacter Infections - pathology Helicobacter pylori Host Response and Inflammation Interferon-gamma - biosynthesis Interleukin-5 - biosynthesis Membrane Glycoproteins - physiology Mice Mice, Inbred C3H Mice, Inbred C57BL Microbiology Pathogenicity, virulence, toxins, bacteriocins, pyrogens, host-bacteria relations, miscellaneous strains
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Zusammenfassung:	Classifications Services IAI Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley Reddit StumbleUpon Twitter current issue Spotlights in the Current Issue IAI About IAI Subscribers Authors Reviewers Advertisers Inquiries from the Press Permissions & Commercial Reprints ASM Journals Public Access Policy Connect to IAI IAI RSS Feeds 1752 N Street N.W. • Washington DC 20036 202.737.3600 • 202.942.9355 fax • journals@asmusa.org Print ISSN: 0019-9567 Online ISSN: 1098-5522 Copyright © 2014 by the American Society for Microbiology. For an alternate route to IAI .asm.org, visit: IAI
ISSN:	0019-9567 1098-5522
DOI:	10.1128/IAI.70.5.2591-2597.2002