발열이 있는 급성신우신염에 동반된 저인산염혈증에 관한 임상적 관찰

Hypophosphatemia is not uncommonly observed in infectious illness and thought to be caused by shift of extracelluar phosphate into cell. Transcellular shift of phosphste is supposed to result from respiratory alkalosis and be augumented by intravenous administration of glucose in some cases. Recently hypophosphatemia was commonly ob- served in malignant neoplastic patients who were re- ceiving intreleukin 2 or tumor necrosis factor which are also known to be important effectors in septice- mia. We tried to evaluate the role of respitory alkalosis, administration of glucose and administration of ibuprofen in hypophosphatemia in infectious illness by measuring plasma P, Ca concentration, arterial blood pH, PcoFEp at admission, plasma P concen- tration after glucose administration and plasma P concentration 90 mimutes after oral administration of 200mg brufen in patients with febrile acute pyelo- nephritis Obtained results were as follows 1) Plasma P concentration of 34 patients at admission were 2.36 ± 0.79mg / dl and hypopho- sphatemia(