Delayed Neurologic Sequelae Caused By Carbon Monoxide Poisoning in a Kidney Transplant Recipient

Diseases that cause significant brain dysfunction include dementia, cerebrovascular disease, and traumatic brain disease. However, in kidney trans - plant recipients, differential diagnoses include calcineurin inhibitor (CNI) encephalopathy,1 posterior reversible encephalopathy syndrome,2 posttransp...

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Veröffentlicht in:Experimental and clinical transplantation 2019-12, Vol.17 (6), p.849-851
Hauptverfasser: Tsuda, Sotaro, Uchida, Junji, Iwai, Tomoaki, Morooka, Chiaki, Kabei, Kazuya, Mino, Toshikazu, Kuwabara, Nobuyuki, Nakatani, Tatsuya
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Sprache:eng
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Zusammenfassung:Diseases that cause significant brain dysfunction include dementia, cerebrovascular disease, and traumatic brain disease. However, in kidney trans - plant recipients, differential diagnoses include calcineurin inhibitor (CNI) encephalopathy,1 posterior reversible encephalopathy syndrome,2 posttransplant lymphoproliferative disorders,3 and progressive multifocal leukoencephalopathy.4 Delayed neurologic sequelae are known to occur after an approximately 40-day asymptomatic period following acute carbon monoxide poisoning,5 but this manifestation has not yet been reported in a kidney transplant recipient. Here, we describe a case of delayed neurologic sequelae after carbon monoxide poisoning in a kidney transplant recipient.
ISSN:1304-0855
2146-8427
DOI:10.6002/ect.2019.0034