Delayed Neurologic Sequelae Caused By Carbon Monoxide Poisoning in a Kidney Transplant Recipient
Diseases that cause significant brain dysfunction include dementia, cerebrovascular disease, and traumatic brain disease. However, in kidney trans - plant recipients, differential diagnoses include calcineurin inhibitor (CNI) encephalopathy,1 posterior reversible encephalopathy syndrome,2 posttransp...
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Veröffentlicht in: | Experimental and clinical transplantation 2019-12, Vol.17 (6), p.849-851 |
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Sprache: | eng |
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Zusammenfassung: | Diseases that cause significant brain dysfunction
include dementia, cerebrovascular disease, and
traumatic brain disease. However, in kidney trans -
plant recipients, differential diagnoses include
calcineurin inhibitor (CNI) encephalopathy,1 posterior
reversible encephalopathy syndrome,2 posttransplant
lymphoproliferative disorders,3 and progressive
multifocal leukoencephalopathy.4 Delayed neurologic
sequelae are known to occur after an approximately
40-day asymptomatic period following acute carbon
monoxide poisoning,5 but this manifestation has not
yet been reported in a kidney transplant recipient.
Here, we describe a case of delayed neurologic
sequelae after carbon monoxide poisoning in a
kidney transplant recipient. |
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ISSN: | 1304-0855 2146-8427 |
DOI: | 10.6002/ect.2019.0034 |