The Interaction Between Pathogens and the Host Coagulation System
Hongmin Sun Life Sciences Institute, University of Michigan, Ann Arbor, Michigan hongmin{at}umich.edu There is mounting evidence that the hemostatic system is critical in host responses to bacterial infection. Invasive bacteria have evolved virulence strategies to interact with host hemostatic facto...
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| Veröffentlicht in: | Physiology (Bethesda, Md.) Md.), 2006-08, Vol.21 (4), p.281-288 |
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| container_title | Physiology (Bethesda, Md.) |
| container_volume | 21 |
| creator | Sun, Hongmin |
| description | Hongmin Sun
Life Sciences Institute, University of Michigan, Ann Arbor, Michigan hongmin{at}umich.edu
There is mounting evidence that the hemostatic system is critical in host responses to bacterial infection. Invasive bacteria have evolved virulence strategies to interact with host hemostatic factors such as plasminogen and fibrinogen for infection. Furthermore, genetic variations in host hemostatic factors also influence host response to bacterial infection. |
| doi_str_mv | 10.1152/physiol.00059.2005 |
| format | Article |
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Life Sciences Institute, University of Michigan, Ann Arbor, Michigan hongmin{at}umich.edu
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Life Sciences Institute, University of Michigan, Ann Arbor, Michigan hongmin{at}umich.edu
There is mounting evidence that the hemostatic system is critical in host responses to bacterial infection. Invasive bacteria have evolved virulence strategies to interact with host hemostatic factors such as plasminogen and fibrinogen for infection. Furthermore, genetic variations in host hemostatic factors also influence host response to bacterial infection.</description><subject>Animals</subject><subject>Bacterial Infections - physiopathology</subject><subject>Blood Coagulation - genetics</subject><subject>Blood Coagulation - physiology</subject><subject>Blood-Borne Pathogens</subject><subject>Fibrinogen - physiology</subject><subject>Genetic Variation</subject><subject>Hemostasis - physiology</subject><subject>Humans</subject><subject>Plasminogen - physiology</subject><subject>Thrombosis - physiopathology</subject><issn>1548-9213</issn><issn>1548-9221</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2006</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNp1kMtOwzAQRS0EoqXwAyxQVohNix9xk-woFaWVKoFEWVvTZNIEpXaIHZX8PekL2LCxrdG5V-NDyDWjA8Ykvy-zxuamGFBKZTTg7XlCukz6YT_inJ3-vJnokAtrPygVMpT8nHTYMByGggVdMlpk6M20wwpilxvtPaLbIGrvFVxmVqitBzrxXEtNjXXe2MCqLmCHvjXW4fqSnKVQWLw63D3yPnlajKf9-cvzbDya92NfBq4f8ygdSrYEH5fMFz7wSNAAoiiBgHGgEUoaQSAkpIwlLN7OcJgAlbCLiB653feWlfms0Tq1zm2MRQEaTW1V-6VAci5akO_BuDLWVpiqssrXUDWKUbUVpw7i1E6c2oprQzeH9nq5xuQ3cjDVAnd7IMtX2Sav8NhiVo3SeWkVZ8pXPNxu-vA_OqmLYoFf7s_M6CLXeIyrMknFN66FkeI</recordid><startdate>20060801</startdate><enddate>20060801</enddate><creator>Sun, Hongmin</creator><general>Am Physiological Soc</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>7X8</scope></search><sort><creationdate>20060801</creationdate><title>The Interaction Between Pathogens and the Host Coagulation System</title><author>Sun, Hongmin</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c457t-c29f651ba4eb1434a29307a99da712a09e509a735af11d1c12a0e6da05aa4eb13</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2006</creationdate><topic>Animals</topic><topic>Bacterial Infections - physiopathology</topic><topic>Blood Coagulation - genetics</topic><topic>Blood Coagulation - physiology</topic><topic>Blood-Borne Pathogens</topic><topic>Fibrinogen - physiology</topic><topic>Genetic Variation</topic><topic>Hemostasis - physiology</topic><topic>Humans</topic><topic>Plasminogen - physiology</topic><topic>Thrombosis - physiopathology</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Sun, Hongmin</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Physiology (Bethesda, Md.)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Sun, Hongmin</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>The Interaction Between Pathogens and the Host Coagulation System</atitle><jtitle>Physiology (Bethesda, Md.)</jtitle><addtitle>Physiology (Bethesda)</addtitle><date>2006-08-01</date><risdate>2006</risdate><volume>21</volume><issue>4</issue><spage>281</spage><epage>288</epage><pages>281-288</pages><issn>1548-9213</issn><eissn>1548-9221</eissn><abstract>Hongmin Sun
Life Sciences Institute, University of Michigan, Ann Arbor, Michigan hongmin{at}umich.edu
There is mounting evidence that the hemostatic system is critical in host responses to bacterial infection. Invasive bacteria have evolved virulence strategies to interact with host hemostatic factors such as plasminogen and fibrinogen for infection. Furthermore, genetic variations in host hemostatic factors also influence host response to bacterial infection.</abstract><cop>United States</cop><pub>Am Physiological Soc</pub><pmid>16868317</pmid><doi>10.1152/physiol.00059.2005</doi><tpages>8</tpages></addata></record> |
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| ispartof | Physiology (Bethesda, Md.), 2006-08, Vol.21 (4), p.281-288 |
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| language | eng |
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| source | MEDLINE; American Physiological Society Paid |
| subjects | Animals Bacterial Infections - physiopathology Blood Coagulation - genetics Blood Coagulation - physiology Blood-Borne Pathogens Fibrinogen - physiology Genetic Variation Hemostasis - physiology Humans Plasminogen - physiology Thrombosis - physiopathology |
| title | The Interaction Between Pathogens and the Host Coagulation System |
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