A bioinformatic and mechanistic study elicits the antifibrotic effect of ursolic acid through the attenuation of oxidative stress with the involvement of ERK, PI3K/Akt, and p38 MAPK signaling pathways in human hepatic stellate cells and rat liver

A bioinformatic and mechanistic study elicits the antifibrotic effect of ursolic acid through the attenuation of oxidative stress with the involvement of ERK, PI3K/Akt, and p38 MAPK signaling pathways in human hepatic stellate cells and rat liver

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Veröffentlicht in:Drug design, development and therapy development and therapy, 2015-01, Vol.9, p.4061
Hauptverfasser: He, Wenhua, Shi, Feng, Zhou, Zhi-Wei, Li, Bimin, Zhang, Kunhe, Zhang, Xinhua, Ouyang, Canhui, Zhou, Shu-Feng, Zhu, Xuan
Format: Artikel
Sprache:eng
Schlagworte:
Cellular proteins
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ISSN:1177-8881
1177-8881