Effect of Inflammatory and Noninflammatory Stress on Beta-Hydroxybutyrate and Free Fatty Acids in Rat Blood
Beta-Hydroxybutyrate and free fatty acid concentrations in rat blood were measured during a variety of inflammatory and noninflammatory stresses. Inflammatory stresses included infection with Streptococcus pneumoniae, endotoxemia and induced turpentine abscess. Noninflammatory stresses included fast...
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creator | Neufeld,Harold A Kaminski,Mitchell V , Jr Pace,Judith A |
description | Beta-Hydroxybutyrate and free fatty acid concentrations in rat blood were measured during a variety of inflammatory and noninflammatory stresses. Inflammatory stresses included infection with Streptococcus pneumoniae, endotoxemia and induced turpentine abscess. Noninflammatory stresses included fasting plus screen-restraint and fasting plus femoral fracture. Inflammatory stresses caused a marked inhibition of the normal fasting-induced ketosis and a reduction in the level of circulating free fatty acids. Noninflammatory stresses caused no inhibition of the normal fasting-induced ketosis but did cause a reduction in the level of circulating free fatty acids. (Author) |
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Inflammatory stresses included infection with Streptococcus pneumoniae, endotoxemia and induced turpentine abscess. Noninflammatory stresses included fasting plus screen-restraint and fasting plus femoral fracture. Inflammatory stresses caused a marked inhibition of the normal fasting-induced ketosis and a reduction in the level of circulating free fatty acids. Noninflammatory stresses caused no inhibition of the normal fasting-induced ketosis but did cause a reduction in the level of circulating free fatty acids. 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Inflammatory stresses included infection with Streptococcus pneumoniae, endotoxemia and induced turpentine abscess. Noninflammatory stresses included fasting plus screen-restraint and fasting plus femoral fracture. Inflammatory stresses caused a marked inhibition of the normal fasting-induced ketosis and a reduction in the level of circulating free fatty acids. Noninflammatory stresses caused no inhibition of the normal fasting-induced ketosis but did cause a reduction in the level of circulating free fatty acids. (Author)</description><subject>Beta-hydroxybutyric acid</subject><subject>BLOOD ANALYSIS</subject><subject>FATTY ACIDS</subject><subject>KETONES</subject><subject>Ketosis</subject><subject>MALES</subject><subject>RATS</subject><subject>Stress Physiology</subject><fulltext>true</fulltext><rsrctype>report</rsrctype><creationdate>1976</creationdate><recordtype>report</recordtype><sourceid>1RU</sourceid><recordid>eNqFi7EKwjAQQLM4iPoHDvcDhVJBXFttqIuDupczuUBomoPkBPP3gji4OT14j7dUU-8cGQF2cI4u4DyjcCqA0cKFo_91N0mUM3CEjgSrodjEr_J4Skko9Fl0IgKNIgVa420GH-GKAl1gtmu1cBgybb5cqa3u78ehsuLNmMVHkrE9tXVz2NfN7k9-AwHtPbg</recordid><startdate>19760715</startdate><enddate>19760715</enddate><creator>Neufeld,Harold A</creator><creator>Kaminski,Mitchell V , Jr</creator><creator>Pace,Judith A</creator><scope>1RU</scope><scope>BHM</scope></search><sort><creationdate>19760715</creationdate><title>Effect of Inflammatory and Noninflammatory Stress on Beta-Hydroxybutyrate and Free Fatty Acids in Rat Blood</title><author>Neufeld,Harold A ; Kaminski,Mitchell V , Jr ; Pace,Judith A</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-dtic_stinet_ADA0286023</frbrgroupid><rsrctype>reports</rsrctype><prefilter>reports</prefilter><language>eng</language><creationdate>1976</creationdate><topic>Beta-hydroxybutyric acid</topic><topic>BLOOD ANALYSIS</topic><topic>FATTY ACIDS</topic><topic>KETONES</topic><topic>Ketosis</topic><topic>MALES</topic><topic>RATS</topic><topic>Stress Physiology</topic><toplevel>online_resources</toplevel><creatorcontrib>Neufeld,Harold A</creatorcontrib><creatorcontrib>Kaminski,Mitchell V , Jr</creatorcontrib><creatorcontrib>Pace,Judith A</creatorcontrib><creatorcontrib>ARMY MEDICAL RESEARCH INST OF INFECTIOUS DISEASES FREDERICK MD</creatorcontrib><collection>DTIC Technical Reports</collection><collection>DTIC STINET</collection></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext_linktorsrc</fulltext></delivery><addata><au>Neufeld,Harold A</au><au>Kaminski,Mitchell V , Jr</au><au>Pace,Judith A</au><aucorp>ARMY MEDICAL RESEARCH INST OF INFECTIOUS DISEASES FREDERICK MD</aucorp><format>book</format><genre>unknown</genre><ristype>RPRT</ristype><btitle>Effect of Inflammatory and Noninflammatory Stress on Beta-Hydroxybutyrate and Free Fatty Acids in Rat Blood</btitle><date>1976-07-15</date><risdate>1976</risdate><abstract>Beta-Hydroxybutyrate and free fatty acid concentrations in rat blood were measured during a variety of inflammatory and noninflammatory stresses. Inflammatory stresses included infection with Streptococcus pneumoniae, endotoxemia and induced turpentine abscess. Noninflammatory stresses included fasting plus screen-restraint and fasting plus femoral fracture. Inflammatory stresses caused a marked inhibition of the normal fasting-induced ketosis and a reduction in the level of circulating free fatty acids. Noninflammatory stresses caused no inhibition of the normal fasting-induced ketosis but did cause a reduction in the level of circulating free fatty acids. (Author)</abstract><oa>free_for_read</oa></addata></record> |
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subjects | Beta-hydroxybutyric acid BLOOD ANALYSIS FATTY ACIDS KETONES Ketosis MALES RATS Stress Physiology |
title | Effect of Inflammatory and Noninflammatory Stress on Beta-Hydroxybutyrate and Free Fatty Acids in Rat Blood |
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