SARS-CoV-2 Pathogenesis: Imbalance in the Renin-Angiotensin System Favors Lung Fibrosis

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Bibliographische Detailangaben
Veröffentlicht in:	Frontiers in cellular and infection microbiology 2020-06, Vol.10, p.340-340
Hauptverfasser:	Delpino, M Victoria, Quarleri, Jorge
Format:	Artikel
Sprache:	eng
Schlagworte:	ACE2 Alveolar Epithelial Cells - enzymology ANG1-7 Angiotensin I - metabolism Angiotensin I - pharmacology Angiotensin II - metabolism Angiotensin-Converting Enzyme 2 Betacoronavirus - pathogenicity Betacoronavirus - physiology Cellular and Infection Microbiology Coronavirus Infections - complications Coronavirus Infections - physiopathology Coronavirus Infections - virology COVID-19 Fibrosis Host Microbial Interactions Humans Lung - enzymology Lung - physiopathology Lung - virology Pandemics Peptide Fragments - metabolism Peptide Fragments - pharmacology Peptidyl-Dipeptidase A - genetics Peptidyl-Dipeptidase A - metabolism Pneumonia, Viral - complications Pneumonia, Viral - physiopathology Pneumonia, Viral - virology Pulmonary Fibrosis - etiology Receptors, Coronavirus Receptors, Virus - genetics Receptors, Virus - metabolism Renin angiotensin system (RAS) Renin-Angiotensin System - physiology SARS-CoV-2 Transforming Growth Factor beta - antagonists & inhibitors Transforming Growth Factor beta - metabolism
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