HTAP2 multi-model estimates of premature human mortality due to intercontinental transport of air pollution and emission sectors

Ambient air pollution from ozone and fine particulate matter is associated with premature mortality. As emissions from one continent influence air quality over others, changes in emissions can also influence human health on other continents. We estimate global air-pollution-related premature mortali...

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Veröffentlicht in:Atmospheric chemistry and physics 2018-07, Vol.18 (14), p.10497-10520
Hauptverfasser: Liang, Ciao-Kai, West, J. Jason, Silva, Raquel A, Bian, Huisheng, Chin, Mian, Davila, Yanko, Dentener, Frank J, Emmons, Louisa, Flemming, Johannes, Folberth, Gerd, Henze, Daven, Im, Ulas, Jonson, Jan Eiof, Keating, Terry J, Kucsera, Tom, Lenzen, Allen, Lin, Meiyun, Lund, Marianne Tronstad, Pan, Xiaohua, Park, Rokjin J, Pierce, R. Bradley, Sekiya, Takashi, Sudo, Kengo, Takemura, Toshihiko
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Sprache:eng
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Zusammenfassung:Ambient air pollution from ozone and fine particulate matter is associated with premature mortality. As emissions from one continent influence air quality over others, changes in emissions can also influence human health on other continents. We estimate global air-pollution-related premature mortality from exposure to PM2.5 and ozone and the avoided deaths due to 20 % anthropogenic emission reductions from six source regions, North America (NAM), Europe (EUR), South Asia (SAS), East Asia (EAS), Russia–Belarus–Ukraine (RBU), and the Middle East (MDE), three global emission sectors, power and industry (PIN), ground transportation (TRN), and residential (RES), and one global domain (GLO), using an ensemble of global chemical transport model simulations coordinated by the second phase of the Task Force on Hemispheric Transport of Air Pollutants (TF HTAP2), and epidemiologically derived concentration response functions. We build on results from previous studies of TF HTAP by using improved atmospheric models driven by new estimates of 2010 anthropogenic emissions (excluding methane), with more source and receptor regions, new consideration of source sector impacts, and new epidemiological mortality functions. We estimate 290 000 (95 % confidence interval (CI): 30 000, 600 000) premature O3-related deaths and 2.8 million (0.5 million, 4.6 million) PM2.5-related premature deaths globally for the baseline year 2010. While 20 % emission reductions from one region generally lead to more avoided deaths within the source region than outside, reducing emissions from MDE and RBU can avoid more O3-related deaths outside of these regions than within, and reducing MDE emissions also avoids more PM2.5-related deaths outside of MDE than within. Our findings that most avoided O3-related deaths from emission reductions in NAM and EUR occur outside of those regions contrast with those of previous studies, while estimates of PM2.5-related deaths from NAM, EUR, SAS, and EAS emission reductions agree well. In addition, EUR, MDE, and RBU have more avoided O3-related deaths from reducing foreign emissions than from domestic reductions. For six regional emission reductions, the total avoided extra-regional mortality is estimated as 6000 (−3400, 15 500) deaths per year and 25 100 (8200, 35 800) deaths per year through changes in O3 and PM2.5, respectively. Interregional transport of air pollutants leads to more deaths through changes in PM2.5 than in O3, even though O3 is transported
ISSN:1680-7324
1680-7316
1680-7324
DOI:10.5194/acp-18-10497-2018