Cell‐autonomous defects contribute to insulin resistance in skeletal muscle

Cell‐autonomous defects contribute to insulin resistance in skeletal muscle

Major pathogenesis and signaling underlying cell‐autonomous muscle insulin resistance in type2 diabetes.

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Bibliographische Detailangaben
Veröffentlicht in:Journal of Cutaneous Immunology and Allergy 2021-07, Vol.12 (7), p.1136-1137
Hauptverfasser: Fujita, Yoshihito, Inagaki, Nobuya
Format: Artikel
Sprache:eng
Schlagworte:
AKT protein
Cell differentiation
Chromatin remodeling
Commentaries
Cytoskeleton
Diabetes
Diabetes mellitus
Gene expression
Glucose
Insulin
Insulin resistance
Kinases
Mitochondria
Muscles
Musculoskeletal system
Myoblasts
Pathogenesis
Phosphorylation
Pluripotency
Protein transport
Proteins
Rapamycin
Respiration
Signal transduction
Skeletal muscle
Stem cells
TOR protein
Transcription
Type 2 diabetes
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Beschreibung
Zusammenfassung:Major pathogenesis and signaling underlying cell‐autonomous muscle insulin resistance in type2 diabetes.
ISSN:2040-1116
2040-1124
DOI:10.1111/jdi.13557