Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation-2

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation-2

Copyright information:Taken from "Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation"BMC Neuroscience 2006;7():55-55.Published online 14 Jul 2006PMCID:PMC1540436.istochemistry with a specific antibody to the...

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Hauptverfasser: Leinhase, Iris, V Michael Holers, Thurman, Joshua M, Harhausen, Denise, Schmidt, Oliver I, Pietzcker, Malte, Mohy E Taha, Rittirsch, Daniel, Huber-Lang, Markus, Smith, Wade R, Ward, Peter A, Stahel, Philip F
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creator Leinhase, Iris
V Michael Holers
Thurman, Joshua M
Harhausen, Denise
Schmidt, Oliver I
Pietzcker, Malte
Mohy E Taha
Rittirsch, Daniel
Huber-Lang, Markus
Smith, Wade R
Ward, Peter A
Stahel, Philip F
description Copyright information:Taken from "Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation"BMC Neuroscience 2006;7():55-55.Published online 14 Jul 2006PMCID:PMC1540436.istochemistry with a specific antibody to the neuronal marker NeuN () or by TUNEL-histochemistry of adjacent sections (). The overall cellular morphology of the TUNEL sections is revealed by DAPI nuclear stain (). The panels represent a 4-fold magnification of the respective panels . The evident destruction of the cortical tissue in brain-injured wild-type mice (upper panels) appears to be structurally preserved in -/- mice (bottom panels). The data shown here are highly reproducible in all tissue sections and animals assessed. Original magnifications: 100× (), 400× ().
doi_str_mv 10.6084/m9.figshare.42317
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title Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation-2
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