Additional file 14 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model
Additional file 14: Supplementary dataset 3. GO analysis of protein dysregulation in the hippocampus due to disease (APP/PS1 mice compared to wildtype controls - APP VEH vs. WT VEH); significantly up regulated group.
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creator | de Veij Mestdagh, Christina F. Koopmans, Frank Breiter, Jonathan C. Timmerman, Jaap A. Vogelaar, Pieter C. Krenning, Guido Mansvelder, Huibert D. Smit, August B. Henning, Robert H. van Kesteren, Ronald E. |
description | Additional file 14: Supplementary dataset 3. GO analysis of protein dysregulation in the hippocampus due to disease (APP/PS1 mice compared to wildtype controls - APP VEH vs. WT VEH); significantly up regulated group. |
doi_str_mv | 10.6084/m9.figshare.22611835 |
format | Dataset |
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GO analysis of protein dysregulation in the hippocampus due to disease (APP/PS1 mice compared to wildtype controls - APP VEH vs. WT VEH); significantly up regulated group.</abstract><pub>figshare</pub><doi>10.6084/m9.figshare.22611835</doi><oa>free_for_read</oa></addata></record> |
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subjects | Biochemistry Cell Biology Chemical Sciences not elsewhere classified Computational Biology FOS: Biological sciences FOS: Chemical sciences Neuroscience Physiology |
title | Additional file 14 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model |
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