Additional file 14 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model

Additional file 14 of The hibernation-derived compound SUL-138 shifts the mitochondrial proteome towards fatty acid metabolism and prevents cognitive decline and amyloid plaque formation in an Alzheimer’s disease mouse model

Additional file 14: Supplementary dataset 3. GO analysis of protein dysregulation in the hippocampus due to disease (APP/PS1 mice compared to wildtype controls - APP VEH vs. WT VEH); significantly up regulated group.

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Bibliographische Detailangaben
Hauptverfasser: de Veij Mestdagh, Christina F., Koopmans, Frank, Breiter, Jonathan C., Timmerman, Jaap A., Vogelaar, Pieter C., Krenning, Guido, Mansvelder, Huibert D., Smit, August B., Henning, Robert H., van Kesteren, Ronald E.
Format: Dataset
Sprache:eng
Schlagworte:
Biochemistry
Cell Biology
Chemical Sciences not elsewhere classified
Computational Biology
FOS: Biological sciences
FOS: Chemical sciences
Neuroscience
Physiology
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Beschreibung
Zusammenfassung:Additional file 14: Supplementary dataset 3. GO analysis of protein dysregulation in the hippocampus due to disease (APP/PS1 mice compared to wildtype controls - APP VEH vs. WT VEH); significantly up regulated group.
DOI:10.6084/m9.figshare.22611835