Additional file 7 of PM2.5 promotes NSCLC carcinogenesis through translationally and transcriptionally activating DLAT-mediated glycolysis reprograming

Additional file 7: Fig. S7. Clinical significance of DLAT expression in NSCLC in TCGA datasets. (A) DLAT was up-regulated in tumor tissues of LUAD (lung adenocarcinoma). (B) The expression level of DLAT in LUSC (lung squamous cell carcinoma) tissue was higher than that in normal tissues. (C) The expression of DLAT protein in LUAD was higher than that in normal tissues. (D) Higher expression level of DLAT in tumor tissues was associated with worse overall survival of NSCLC. (E) Higher expression level of DLAT in tumor tissues was associated with worse progression free survival of NSCLC patients. (F) DLAT was up-regulated in other types of primary tumors.