Endorphin mediation of mesenteric blood flow after endotoxemia in sheep

CONCLUSIONSThe decrease in cardiac output noted with endotoxin can be accounted for by the decrease in the blood flow in the cephalic mesenteric artery. This phenomenon can be attributed, at least in part, to the release of endorphins. There is both a vasodilation and constriction during endotoxemia...

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Veröffentlicht in:	Critical care medicine 1992-03, Vol.20 (3), p.402-408
Hauptverfasser:	NAVARATNAM, NEVILLE, HERNDON, DAVID N, WOODSON, LEE C, LINARES, HUGO A, MORRIS, STEPHEN, TRABER, DANIEL L
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Bacterial diseases beta-Endorphin - blood Biological and medical sciences Blood Pressure - drug effects Cardiac Output - drug effects Endotoxins - antagonists & inhibitors Endotoxins - toxicity Escherichia coli Experimental bacterial diseases and models Female Infectious diseases Medical sciences Naloxone - pharmacology Radioimmunoassay Sheep Splanchnic Circulation - drug effects
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Zusammenfassung:	CONCLUSIONSThe decrease in cardiac output noted with endotoxin can be accounted for by the decrease in the blood flow in the cephalic mesenteric artery. This phenomenon can be attributed, at least in part, to the release of endorphins. There is both a vasodilation and constriction during endotoxemia in the ovine model. (Crit Care Med 1992; 20:402–408)
ISSN:	0090-3493 1530-0293
DOI:	10.1097/00003246-199203000-00018