Liver function during chronic renal failure in rabbits
In rabbits with chronic renal insufficiency the prothrombin index was increased by 25% and the alanine aminotransferase activity decreased by 20%; the results of other routine tests of hepatic function were not affected. The galactose elimination capacity was decreased by 12%, whereas the body clear...
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| Veröffentlicht in: | Experientia 1984-11, Vol.40 (11), p.1255-1256 |
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| creator | TVEDEGAARD, E POULSEN, H. E VILSTRUP, H THOMSEN, H. K |
| description | In rabbits with chronic renal insufficiency the prothrombin index was increased by 25% and the alanine aminotransferase activity decreased by 20%; the results of other routine tests of hepatic function were not affected. The galactose elimination capacity was decreased by 12%, whereas the body clearance of antipyrine was unchanged. No change in hepatocytic structure was found. |
| doi_str_mv | 10.1007/BF01946660 |
| format | Article |
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E ; VILSTRUP, H ; THOMSEN, H. K</creator><creatorcontrib>TVEDEGAARD, E ; POULSEN, H. E ; VILSTRUP, H ; THOMSEN, H. K</creatorcontrib><description>In rabbits with chronic renal insufficiency the prothrombin index was increased by 25% and the alanine aminotransferase activity decreased by 20%; the results of other routine tests of hepatic function were not affected. The galactose elimination capacity was decreased by 12%, whereas the body clearance of antipyrine was unchanged. No change in hepatocytic structure was found.</description><identifier>ISSN: 0014-4754</identifier><identifier>EISSN: 1420-9071</identifier><identifier>DOI: 10.1007/BF01946660</identifier><identifier>PMID: 6500010</identifier><identifier>CODEN: EXPEAM</identifier><language>eng</language><publisher>Basel: Birkhäuser</publisher><subject>Alanine Transaminase - metabolism ; Alkaline Phosphatase - metabolism ; Animals ; Biological and medical sciences ; Galactose - metabolism ; Kidney Failure, Chronic - pathology ; Kidney Failure, Chronic - physiopathology ; Liver - pathology ; Liver - physiopathology ; Male ; Medical sciences ; Nephrology. Urinary tract diseases ; Nephropathies. Renovascular diseases. 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No change in hepatocytic structure was found.</description><subject>Alanine Transaminase - metabolism</subject><subject>Alkaline Phosphatase - metabolism</subject><subject>Animals</subject><subject>Biological and medical sciences</subject><subject>Galactose - metabolism</subject><subject>Kidney Failure, Chronic - pathology</subject><subject>Kidney Failure, Chronic - physiopathology</subject><subject>Liver - pathology</subject><subject>Liver - physiopathology</subject><subject>Male</subject><subject>Medical sciences</subject><subject>Nephrology. Urinary tract diseases</subject><subject>Nephropathies. Renovascular diseases. Renal failure</subject><subject>Prothrombin Time</subject><subject>Rabbits</subject><subject>Renal failure</subject><issn>0014-4754</issn><issn>1420-9071</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>1984</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><recordid>eNpFkEFLwzAYhoMoc04v3oUcxINQ_dKkSXrU4VQYeNFzSdJEI106k1bw3xtZmafv8Dy8vN-L0DmBGwIgbu9XQGrGOYcDNCeshKIGQQ7RHICwgomKHaOTlD4BKFSEz9CMV5AZzBFf-28bsRuDGXwfcDtGH96x-Yh98AZHG1SHnfLdGC32AUeltR_SKTpyqkv2bLoL9LZ6eF0-FeuXx-fl3bowlJChUIZzxiopoW4JdSVVjBPQwpm2FZZJyzTLzUvRVlpryYXhqs5A8QxAUbpAV7vcbey_RpuGZuOTsV2ngu3H1EhC8yeCZ_F6J5rYpxSta7bRb1T8aQg0fyM1_yNl-WJKHfXGtnt1WiXzy4mrZFTnogrGp70m66qStKS_orRsWQ</recordid><startdate>19841115</startdate><enddate>19841115</enddate><creator>TVEDEGAARD, E</creator><creator>POULSEN, H. E</creator><creator>VILSTRUP, H</creator><creator>THOMSEN, H. 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K</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c311t-ac664458809d13f23a4610b7fcdd7e48e4b419427d5bbb867c6a97e4a64b40a33</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>1984</creationdate><topic>Alanine Transaminase - metabolism</topic><topic>Alkaline Phosphatase - metabolism</topic><topic>Animals</topic><topic>Biological and medical sciences</topic><topic>Galactose - metabolism</topic><topic>Kidney Failure, Chronic - pathology</topic><topic>Kidney Failure, Chronic - physiopathology</topic><topic>Liver - pathology</topic><topic>Liver - physiopathology</topic><topic>Male</topic><topic>Medical sciences</topic><topic>Nephrology. Urinary tract diseases</topic><topic>Nephropathies. Renovascular diseases. Renal failure</topic><topic>Prothrombin Time</topic><topic>Rabbits</topic><topic>Renal failure</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>TVEDEGAARD, E</creatorcontrib><creatorcontrib>POULSEN, H. E</creatorcontrib><creatorcontrib>VILSTRUP, H</creatorcontrib><creatorcontrib>THOMSEN, H. K</creatorcontrib><collection>Pascal-Francis</collection><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>MEDLINE - Academic</collection><jtitle>Experientia</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>TVEDEGAARD, E</au><au>POULSEN, H. E</au><au>VILSTRUP, H</au><au>THOMSEN, H. K</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>Liver function during chronic renal failure in rabbits</atitle><jtitle>Experientia</jtitle><addtitle>Experientia</addtitle><date>1984-11-15</date><risdate>1984</risdate><volume>40</volume><issue>11</issue><spage>1255</spage><epage>1256</epage><pages>1255-1256</pages><issn>0014-4754</issn><eissn>1420-9071</eissn><coden>EXPEAM</coden><abstract>In rabbits with chronic renal insufficiency the prothrombin index was increased by 25% and the alanine aminotransferase activity decreased by 20%; the results of other routine tests of hepatic function were not affected. The galactose elimination capacity was decreased by 12%, whereas the body clearance of antipyrine was unchanged. No change in hepatocytic structure was found.</abstract><cop>Basel</cop><cop>Boston, MA</cop><cop>Berlin</cop><pub>Birkhäuser</pub><pmid>6500010</pmid><doi>10.1007/BF01946660</doi><tpages>2</tpages></addata></record> |
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| ispartof | Experientia, 1984-11, Vol.40 (11), p.1255-1256 |
| issn | 0014-4754 1420-9071 |
| language | eng |
| recordid | cdi_crossref_primary_10_1007_BF01946660 |
| source | MEDLINE; 2022 ECC(Springer) |
| subjects | Alanine Transaminase - metabolism Alkaline Phosphatase - metabolism Animals Biological and medical sciences Galactose - metabolism Kidney Failure, Chronic - pathology Kidney Failure, Chronic - physiopathology Liver - pathology Liver - physiopathology Male Medical sciences Nephrology. Urinary tract diseases Nephropathies. Renovascular diseases. Renal failure Prothrombin Time Rabbits Renal failure |
| title | Liver function during chronic renal failure in rabbits |
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