A 2D elastic wall model to analyze the hyper‐viscous effects on blood flow across abdominal aortic aneurysm in COVID patients by fluid–structure interaction technique with ALE formulation

The worldwide dissemination of the coronavirus, as well as its dependencies, is being aided by detrimental urbanization. The impact of the viral infection upon an infected person is also thought to be influenced by any pre‐existing medical issues. Recent investigations have demonstrated that COVID‐19 patients have a higher degree of blood viscosity as a result of alterations in morphology in blood cells. Examining the importance of hyper‐viscosity in patients with COVID is crucial in determining the diseases of the arteries since viscosity is a key flow characteristic that affects the flow across a stenosis as well as an aneurysm. In the current study, a patient‐specific instance with an aneurysm across the abdominal aortal walls was taken into consideration. Given that abdominal aneurysms occur frequently in people, the high viscosity values are taken into account while examining the hyper‐viscous impact of fluid on its passage through the affected aorta. The current study is the initial endeavor to use the Arbitrary Lagrangian–Eulerian (ALE) Technique to study the impact of fluid–structure interaction (FSI) on the movement of fluid across an aneurysmal aortal section in conjunction with hyper‐viscous anomalous blood characteristics. The investigation backs up the different medical results that described the negative consequences of blood hyper‐viscosity. The computational findings from employing a finite element method (FEM) solver to resolve the fluid mechanical computations complemented alongside the solid mechanical principles indicate a possibility that the increased stress imposed by the hyper‐viscous propagates on the interior walls of the aneurysmal aorta may prompt the aneurysmal pouch to grow or breakdown. Also, the hyper‐viscous blood in COVID patients destroys the smooth muscle cells of the media layer endangering its normal structure and functions and leading to the development of new aneurysms.