Photoactive g-C3N4 Nanosheets for Light-Induced Suppression of Alzheimer's β-Amyloid Aggregation and Toxicity

Graphitic carbon nitride (g‐C3N4) has a suppressive capability toward Alzheimer's Aβ aggregation under light‐illumination. Photoinduced electrons of g‐C3N4 generate reactive oxygen resulting in photooxidation of amyloid peptides that blocks Aβ aggregation. Fe doping of g‐C3N4 frameworks results...

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Veröffentlicht in:	Advanced healthcare materials 2016-07, Vol.5 (13), p.1560-1565
Hauptverfasser:	Chung, You Jung, Lee, Byung Il, Ko, Jong Wan, Park, Chan Beum
Format:	Artikel
Sprache:	eng
Schlagworte:	Alzheimer's disease beta-amyloid graphitic carbon nitride nanomaterials photodynamic therapy
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creator	Chung, You Jung Lee, Byung Il Ko, Jong Wan Park, Chan Beum
description	Graphitic carbon nitride (g‐C3N4) has a suppressive capability toward Alzheimer's Aβ aggregation under light‐illumination. Photoinduced electrons of g‐C3N4 generate reactive oxygen resulting in photooxidation of amyloid peptides that blocks Aβ aggregation. Fe doping of g‐C3N4 frameworks results in enhanced optical properties and even stronger inhibition of Aβ aggregation.
doi_str_mv	10.1002/adhm.201500964
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Healthcare Mater</addtitle><date>2016-07-06</date><risdate>2016</risdate><volume>5</volume><issue>13</issue><spage>1560</spage><epage>1565</epage><pages>1560-1565</pages><issn>2192-2640</issn><eissn>2192-2659</eissn><abstract>Graphitic carbon nitride (g‐C3N4) has a suppressive capability toward Alzheimer's Aβ aggregation under light‐illumination. Photoinduced electrons of g‐C3N4 generate reactive oxygen resulting in photooxidation of amyloid peptides that blocks Aβ aggregation. Fe doping of g‐C3N4 frameworks results in enhanced optical properties and even stronger inhibition of Aβ aggregation.</abstract><pub>Blackwell Publishing Ltd</pub><doi>10.1002/adhm.201500964</doi><tpages>6</tpages></addata></record>
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subjects	Alzheimer's disease beta-amyloid graphitic carbon nitride nanomaterials photodynamic therapy
title	Photoactive g-C3N4 Nanosheets for Light-Induced Suppression of Alzheimer's β-Amyloid Aggregation and Toxicity
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