Sequence variants of toll-like receptor 4 are associated with prostate cancer risk: Results from the cancer prostate in sweden study

Inflammation has been implicated as an etiological factor in several human cancers. Growing evidence suggests that chronic inflammation may also play a role in the etiology of prostate cancer. Considering that genetic susceptibility is a major risk factor for this disease, we hypothesize that sequen...

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Veröffentlicht in:	Cancer research (Chicago, Ill.) Ill.), 2004-04, Vol.64 (8), p.2918-2922
Hauptverfasser:	ZHENG, S. Lilly, AUGUSTSSON-BÄLTER, Katarina, ADAMS, Tamara S, MEYERS, Deborah A, ISAACS, William B, JIANFENG XU, GRÖNBERG, Henrik, CHANG, Baoli, HEDELIN, Maria, LIWU LI, ADAMI, Hans-Olov, BENSEN, Jeanette, GE LI, JOHNASSON, Jan-Erik, TURNER, Aubrey R
Format:	Artikel
Sprache:	eng
Schlagworte:	3' Untranslated Regions - genetics Adenocarcinoma - genetics Adenocarcinoma - metabolism Adenocarcinoma - pathology Adult Aged Alleles Antineoplastic agents Biological and medical sciences Case-Control Studies Genetic Predisposition to Disease Humans Male Medical sciences Membrane Glycoproteins - genetics Membrane Glycoproteins - metabolism Middle Aged Neoplasm Staging Pharmacology. Drug treatments Polymorphism, Single Nucleotide Prostatic Neoplasms - genetics Prostatic Neoplasms - metabolism Prostatic Neoplasms - pathology Protein Isoforms Receptors, Cell Surface - genetics Receptors, Cell Surface - metabolism Toll-Like Receptor 4 Toll-Like Receptors Tumors
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