Intermittent Hypoxia causes targeted disruption to NMDA receptor dependent synaptic plasticity in area CA1 of the hippocampus

Changed NMDA receptor (NMDAr) physiology is implicated with cognitive deficit resulting from conditions ranging from normal aging to neurological disease. Using intermittent hypoxia (IH) to experimentally model untreated sleep apnea, a clinical condition whose comorbidities include neurocognitive im...

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Veröffentlicht in:	Experimental neurology 2021-10, Vol.344, p.113808-113808, Article 113808
Hauptverfasser:	Arias-Cavieres, Alejandra, Fonteh, Ateh, Castro-Rivera, Carolina I., Garcia, Alfredo J.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals CA1 Region, Hippocampal - metabolism Cells, Cultured Female Hippocampus Hypoxia, Brain - metabolism Long term depression Long term potentiation Male Mice Mice, Inbred C57BL Neuronal Plasticity - physiology Oxidative stress Oxidative Stress - physiology Receptors, N-Methyl-D-Aspartate - metabolism Sleep apnea Sleep Apnea Syndromes - metabolism
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