Altered p16Ink4a, IL-1β, and Lamin b1 Protein Expression Suggest Cellular Senescence in Deep Endometriotic Lesions

Endometriosis causes immunological and cellular alterations. Endometriosis lesions have lower levels of lamin b1 than the endometrium. Moreover, high levels of pro-inflammatory markers are observed in the peritoneal fluid, follicular fluid, and serum in endometriosis lesions. Thus, we hypothesized t...

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Veröffentlicht in:	International journal of molecular sciences 2022-02, Vol.23 (5), p.2476
Hauptverfasser:	Malvezzi, Helena, Dobo, Cristine, Filippi, Renee Zon, Mendes do Nascimento, Helen, Palmieri da Silva e Sousa, Laura, Meola, Juliana, Piccinato, Carla Azevedo, Podgaec, Sérgio
Format:	Artikel
Sprache:	eng
Schlagworte:	Age Angiogenesis Apoptosis Body mass index Cell cycle Cytokines Disease Endometriosis Endometrium Follicular fluid Growth factors IL-1β Immunology Inflammation INK4a protein Interleukin 4 Laparoscopy Lesions Markers Menstruation Microenvironments p16 Protein Patients Peritoneal fluid Phenotypes Protein expression Proteins Senescence Therapeutic applications Tumor necrosis factor-TNF
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