Serotonin Deficiency Is Associated With Delayed Gastric Emptying

Gastrointestinal (GI) motility is regulated by serotonin (5-hydroxytryptamine [5-HT]), which is primarily produced by enterochromaffin (EC) cells in the GI tract. However, the precise roles of EC cell-derived 5-HT in regulating gastric motility remain a major point of conjecture. Using a novel trans...

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Veröffentlicht in:	Gastroenterology (New York, N.Y. 1943) N.Y. 1943), 2021-06, Vol.160 (7), p.2451-2466.e19
Hauptverfasser:	Wei, Lai, Singh, Rajan, Ha, Se Eun, Martin, Alyce M., Jones, Lauren A., Jin, Byungchang, Jorgensen, Brian G., Zogg, Hannah, Chervo, Tyler, Gottfried-Blackmore, Andres, Nguyen, Linda, Habtezion, Aida, Spencer, Nick J., Keating, Damien J., Sanders, Kenton M., Ro, Seungil
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cell Line Enterochromaffin Cells Enterochromaffin Cells - physiology Gastric Emptying - genetics Gastrointestinal Motility Gastrointestinal Transit - genetics Humans Idiopathic Gastroparesis Mice Mice, Transgenic Serotonin Serotonin - deficiency Slow-Transit Constipation Tryptophan Hydroxylase - metabolism
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