Systemic bone loss following myocardial infarction in mice

Myocardial infarction (MI) and osteoporotic fracture are leading causes of morbidity and mortality, and epidemiological evidence linking their incidence suggests possible crosstalk. MI can exacerbate atherosclerosis through the sympathetic nervous system (SNS) activation and β3 adrenoreceptor‐mediat...

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Veröffentlicht in:	Journal of orthopaedic research 2021-04, Vol.39 (4), p.739-749
Hauptverfasser:	Tjandra, Priscilla M., Paralkar, Manali P., Osipov, Benjamin, Chen, Yi‐Je, Zhao, Fengdong, Ripplinger, Crystal M., Christiansen, Blaine A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Absorptiometry, Photon Animals Body Composition Bone Density Bone Diseases, Metabolic - complications bone loss DXA Femur - pathology Hematopoietic Stem Cells - metabolism Lumbar Vertebrae - pathology Male Mice Mice, Inbred C57BL Monocytes - metabolism myocardial infarction Myocardial Infarction - complications Osteoclasts - metabolism Osteoporotic Fractures - complications Propanolamines - pharmacology Receptors, Adrenergic, beta-3 - metabolism Stress, Mechanical Sympathetic Nervous System X-Ray Microtomography β3‐adrenergic receptor μCT
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