CaMKII inhibitor 1 (CaMK2N1) mRNA is upregulated following LTP induction in hippocampal slices

CaMK2N1 and CaMK2N2 (also known as CaMKIINα and β) are endogenous inhibitors of calcium/calmodulin‐dependent kinase II (CaMKII), an enzyme critical for memory and long‐term potentiation (LTP), a form of synaptic plasticity thought to underlie learning. CaMK2N1/2 mRNAs are rapidly and differentially...

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Veröffentlicht in:	Synapse (New York, N.Y.) N.Y.), 2020-10, Vol.74 (10), p.e22158-n/a
Hauptverfasser:	Astudillo, Daniela, Karmelic, Daniel, Casas, Barbara S., Otmakhov, Nikolai, Palma, Veronica, Sanhueza, Magdalena
Format:	Artikel
Sprache:	eng
Schlagworte:	Amygdala Animals Brain slice preparation Ca2+/calmodulin-dependent protein kinase II Calcium-binding protein Calcium-Binding Proteins - genetics Calcium-Binding Proteins - metabolism CaMK2N1 CaMK2N2 CaMKII Fear conditioning gene expression Hippocampus Hippocampus - metabolism Hippocampus - physiology Kinases Long-Term Potentiation LTP Male mRNA Neuronal Plasticity Rats Rats, Sprague-Dawley RNA, Messenger - genetics RNA, Messenger - metabolism Synaptic plasticity Up-Regulation
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