Epstein–Barr virus peptides derived from latent cycle proteins alter NKG2A + NK cell effector function

Natural killer (NK) cells control viral infection through the interaction between inhibitory receptors and human leukocyte antigen (HLA) ligands and bound peptide. NK cells expressing the inhibitory receptor NKG2A/CD94 recognize and respond to autologous B cells latently infected with Epstein–Barr v...

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Veröffentlicht in:	Scientific reports 2020-11, Vol.10 (1), p.19973-19973, Article 19973
Hauptverfasser:	Mbiribindi, Berenice, Pena, Josselyn K., Arvedson, Matthew P., Moreno Romero, Claudia, McCarthy, Sarah R., Hatton, Olivia L., Esquivel, Carlos O., Martinez, Olivia M., Krams, Sheri M.
Format:	Artikel
Sprache:	eng
Schlagworte:	631/250 631/80 B-Lymphocytes - metabolism Cell activation Cell Line Epstein-Barr virus Herpesvirus 4, Human - metabolism Histocompatibility antigen HLA Histocompatibility Antigens Class I - metabolism Humanities and Social Sciences Humans Killer Cells, Natural - metabolism Latent infection Latent Infection - metabolism Ligands Lymphocytes B multidisciplinary Natural killer cells NK Cell Lectin-Like Receptor Subfamily C - metabolism NKG2 antigen Nucleotide sequence Peptides Peptides - metabolism Proteins Science Science (multidisciplinary)
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