Role of Muscle-Specific Histone Methyltransferase (Smyd1) in Exercise-Induced Cardioprotection against Pathological Remodeling after Myocardial Infarction

Pathological remodeling is the main detrimental complication after myocardial infarction (MI). Overproduction of reactive oxygen species (ROS) in infarcted myocardium may contribute to this process. Adequate exercise training after MI may reduce oxidative stress-induced cardiac tissue damage and rem...

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Veröffentlicht in:	International journal of molecular sciences 2020-10, Vol.21 (19), p.7010
Hauptverfasser:	Liang, Qiaoqin, Cai, Mengxin, Zhang, Jiaqi, Song, Wei, Zhu, Wanyu, Xi, Lei, Tian, Zhenjun
Format:	Artikel
Sprache:	eng
Schlagworte:	Actin Actinin AMP-activated protein kinase Animals Brain natriuretic peptide Cardiac function Cardiac muscle Cardiomyocytes Coronary artery DNA-Binding Proteins - biosynthesis Exercise Fibrosis Fitness training programs Gene Expression Regulation, Developmental Health aspects Heart attack Heart attacks Histone methyltransferase Histones Hsp90 protein Hydrogen peroxide Hypertrophy Ischemia Kinases Male Metabolism Methyltransferases Muscles Myocardial infarction Myocardial Infarction - enzymology Myocardial Infarction - pathology Myocardial Infarction - prevention & control Myocardium Oxidative stress Phosphorylation Physical Conditioning, Animal Physical fitness Physical training Physiological aspects Protein expression Proteins Rats Rats, Sprague-Dawley Reactive oxygen species Resistance training Skeletal muscle Thioredoxins - biosynthesis Treadmills Ventricular Remodeling
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