Ganoderic acid A protects neural cells against NO stress injury in vitro via stimulating β adrenergic receptors

Excessive nitric oxide (NO) causes extensive damage to the nervous system, and the adrenergic system is disordered in many neuropsychiatric diseases. However, the role of the adrenergic system in protection of the nervous system against sodium nitroprusside (SNP) injury remains unclear. In this stud...

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Veröffentlicht in:	Acta pharmacologica Sinica 2020-04, Vol.41 (4), p.516-522
Hauptverfasser:	Yu, Zi-ru, Jia, Wei-hua, Liu, Chao, Wang, Hong-qing, Yang, Hai-guang, He, Guo-rong, Chen, Ruo-yun, Du, Guan-hua
Format:	Artikel
Sprache:	eng
Schlagworte:	Adrenergic receptors Animals Biomedical and Life Sciences Biomedicine Cell culture Cell Survival - drug effects Cytotoxicity Dose-Response Relationship, Drug Epinephrine Ganoderic acid Heptanoic Acids - chemistry Heptanoic Acids - pharmacology Humans Immunology Internal Medicine Lanosterol - analogs & derivatives Lanosterol - chemistry Lanosterol - pharmacology Medical Microbiology Mental disorders Metoprolol Molecular Conformation Nervous system Neurons - drug effects Neuroprotection Neuroprotective Agents - chemistry Neuroprotective Agents - pharmacology Nitric oxide Nitric Oxide - antagonists & inhibitors Nitric Oxide - metabolism Nitroprusside - pharmacology Oxidative Stress - drug effects PC12 Cells Pharmacology/Toxicology Phentolamine Pheochromocytoma cells Rats Receptors, Adrenergic, beta - metabolism Sodium nitroprusside Structure-Activity Relationship Tumor Cells, Cultured Vaccine
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