CYP4F2 and VKORC1 Polymorphisms Amplify the Risk of Carotid Plaque Formation

Atherosclerosis represents the process by which fibrous plaques are formed in the arterial wall, increasing its rigidity with a subsequent decrease in blood flow which can lead to several cardiovascular events. Seeing as vitamin K antagonists are involved in the pathogenesis of atherosclerosis, we d...

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Veröffentlicht in:	Genes 2020-07, Vol.11 (7), p.822
Hauptverfasser:	Vesa, Stefan Cristian, Vlaicu, Sonia Irina, Vacaras, Vitalie, Crisan, Sorin, Sabin, Octavia, Pasca, Sergiu, Trifa, Adrian Pavel, Rusz-Fogarasi, Tamas, Sava, Madalina, Buzoianu, Anca Dana
Format:	Artikel
Sprache:	eng
Schlagworte:	Aged Antagonists Anticoagulants Antihypertensives Arteriosclerosis Blood flow Calcification Cardiac arrhythmia Cardiovascular disease Carotid Artery Diseases - genetics Carotid Artery Diseases - pathology Congestive heart failure Coronary artery disease Cytochrome P450 Family 4 - genetics Deoxyribonucleic acid Diabetes mellitus DNA Dyslipidemia Enzymes Female Femur Fibrillation Health risk assessment Heart failure Humans Hypertension Inflammation Insulin Ischemia Male Metabolism Middle Aged Mutation Obesity Patients Plaque, Atherosclerotic - genetics Plaque, Atherosclerotic - pathology Plaques Polymorphism, Single Nucleotide Proteins Variables Veins & arteries Vitamin K Vitamin K Epoxide Reductases - genetics
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